PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20467441-0	2010	Nitric oxide inhibition of Drp1-mediated mitochondrial fission is critical for myogenic differentiation.	Nitric Oxide	0-12	dynamin 1 like	Homo sapiens	27-31	
19342591-5	2009	We found that nitric oxide produced in response to beta-amyloid protein, thought to be a key mediator of Alzheimer"s disease, triggered mitochondrial fission, synaptic loss, and neuronal damage, in part via S-nitrosylation of dynamin-related protein 1 (forming SNO-Drp1).	Nitric Oxide	14-26	dynamin 1 like	Homo sapiens	226-251	
19342591-5	2009	We found that nitric oxide produced in response to beta-amyloid protein, thought to be a key mediator of Alzheimer"s disease, triggered mitochondrial fission, synaptic loss, and neuronal damage, in part via S-nitrosylation of dynamin-related protein 1 (forming SNO-Drp1).	Nitric Oxide	14-26	dynamin 1 like	Homo sapiens	265-269	
30029585-5	2018	In a recent study, we demonstrated that S-nitrosylation, the reversible modification of cysteine residues by nitric oxide (NO), hyperactivates mitochondrial fragmentation by targeting DNM1L/Drp1 (dynamin 1-like) at Cys644, but inhibits mitophagy, the concomitant occurrence of these conditions driving cell senescence.	Nitric Oxide	109-121	dynamin 1 like	Homo sapiens	184-189	
30029585-5	2018	In a recent study, we demonstrated that S-nitrosylation, the reversible modification of cysteine residues by nitric oxide (NO), hyperactivates mitochondrial fragmentation by targeting DNM1L/Drp1 (dynamin 1-like) at Cys644, but inhibits mitophagy, the concomitant occurrence of these conditions driving cell senescence.	Nitric Oxide	109-121	dynamin 1 like	Homo sapiens	190-194	
19417214-3	2009	Nitric oxide (NO) produced in response to Abeta induces S-nitrosylation of the mitochondrial division protein, dynamin-related protein 1 (Drp-1), which leads to excessive mitochondrial fission, synaptic loss, and neuronal damage.	Nitric Oxide	0-12	dynamin 1 like	Homo sapiens	111-136	
19417214-3	2009	Nitric oxide (NO) produced in response to Abeta induces S-nitrosylation of the mitochondrial division protein, dynamin-related protein 1 (Drp-1), which leads to excessive mitochondrial fission, synaptic loss, and neuronal damage.	Nitric Oxide	0-12	dynamin 1 like	Homo sapiens	138-143