PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14660009-3	2003	The postulated mechanisms of this atheroprotective effect are the antioxidant actions of angiotensin-converting enzyme inhibitors and their enhancement of the endothelial elaboration of bioactive nitric oxide.	Nitric Oxide	196-208	angiotensin I converting enzyme	Bos taurus	89-118	
23729620-0	2013	ACE inhibition enhances bradykinin relaxations through nitric oxide and B1 receptor activation in bovine coronary arteries.	Nitric Oxide	55-67	angiotensin I converting enzyme	Bos taurus	0-3	
16497147-0	2006	Kinin- and angiotensin-converting enzyme (ACE) inhibitor-mediated nitric oxide production in endothelial cells.	Nitric Oxide	66-78	angiotensin I converting enzyme	Bos taurus	11-40	
16497147-0	2006	Kinin- and angiotensin-converting enzyme (ACE) inhibitor-mediated nitric oxide production in endothelial cells.	Nitric Oxide	66-78	angiotensin I converting enzyme	Bos taurus	42-45	
14660009-4	2003	The aim of this study was to assess the comparative effects of three angiotensin-converting enzyme inhibitors on endothelial nitric oxide production and action, and on endothelial oxidative stress.	Nitric Oxide	125-137	angiotensin I converting enzyme	Bos taurus	69-98	
8293767-9	1993	Thus, ACE inhibition causes selective coronary vasodilation by potentiating the bradykinin-induced release of nitric oxide from the endothelium.	Nitric Oxide	110-122	angiotensin I converting enzyme	Bos taurus	6-9