PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32924701-0	2020	Resveratrol prevents TNF-alpha-induced VCAM-1 and ICAM-1 upregulation in endothelial progenitor cells via reduction of NF-kappaB activation.	Resveratrol	0-11	vascular cell adhesion molecule 1	Homo sapiens	39-45	
32924701-5	2020	RESULTS: MNC adhesion to TNF-alpha-treated EPCs and VCAM-1/ICAM-1/E-selectin levels in EPCs were increased following TNF-alpha stimulation and decreased following RSV treatment.	Resveratrol	163-166	vascular cell adhesion molecule 1	Homo sapiens	52-58	
32924701-8	2020	RSV"s mechanism of action might involve downregulation of VCAM-1, ICAM-1 and E-selectin by partial blockade of TNF-alpha-induced NF-kappaB activation and IkappaB-alpha phosphorylation in EPCs.	Resveratrol	0-3	vascular cell adhesion molecule 1	Homo sapiens	58-64	
29208243-8	2017	Resveratrol significantly mitigated tumor necrosis factor-alpha stimulated endothelial expression of ICAM1, VCAM1, E-selectin and endothelin-1 and prevented an increase in endothelial monolayer permeability.	Resveratrol	0-11	vascular cell adhesion molecule 1	Homo sapiens	108-113	
28500309-8	2017	Endothelial dysfunction was induced in HUVECs using TNFalpha, increasing expression of cell adhesion molecule VCAM1 and adhesion of peripheral blood mononuclear cells, both of which were increased further by resveratrol.	Resveratrol	208-219	vascular cell adhesion molecule 1	Homo sapiens	110-115	
20740479-4	2011	RSV and its analogues inhibited, albeit differentially, the protein and mRNA expression levels of inducible cell adhesion molecules, ICAM-1 and VCAM-1, in cultured endothelial cells.	Resveratrol	0-3	vascular cell adhesion molecule 1	Homo sapiens	144-150	
26410779-5	2015	The analysis of pro-inflammatory cytokines release by ELISA revealed that resveratrol, lipoic acid melatonin and Co-Q10 inhibited the BBB endothelial release of pro-inflammatory cytokines IL-6 and IL-8, reduced (not Co-Q10) CSE-induced up-regulation of Platelet Cell Adhesion Molecule-1 (PECAM-1), Vascular Cell Adhesion Molecule-1 (VCAM-1) & E-selectin and inhibited monocytes-endothelial cell adhesion.	Resveratrol	74-85	vascular cell adhesion molecule 1	Homo sapiens	298-331	
26410779-5	2015	The analysis of pro-inflammatory cytokines release by ELISA revealed that resveratrol, lipoic acid melatonin and Co-Q10 inhibited the BBB endothelial release of pro-inflammatory cytokines IL-6 and IL-8, reduced (not Co-Q10) CSE-induced up-regulation of Platelet Cell Adhesion Molecule-1 (PECAM-1), Vascular Cell Adhesion Molecule-1 (VCAM-1) & E-selectin and inhibited monocytes-endothelial cell adhesion.	Resveratrol	74-85	vascular cell adhesion molecule 1	Homo sapiens	333-339	
9846848-4	1998	DESIGN: Because resveratrol is a tyrosine kinase inhibitor like other members of the tyrphostin family, we hypothesized that it has the ability to modify intracellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) expression by stimulated endothelial cells.	Resveratrol	16-27	vascular cell adhesion molecule 1	Homo sapiens	201-234	
19481207-11	2009	Resveratrol, the active component of red wine, inhibited the expression of both VCAM-1 and E-selectin, and reduced monocytic cell recruitment by 50% at 20 micromol/L.	Resveratrol	0-11	vascular cell adhesion molecule 1	Homo sapiens	80-86	
17586618-8	2007	Finally, we found that nutritionally relevant concentrations of RSV and HT, but not folate and vitamin B6, reduce (by >60% at 10(-6) mol/l) Hcy-induced VCAM-1 expression and monocytoid cell adhesion to the endothelium.	Resveratrol	64-67	vascular cell adhesion molecule 1	Homo sapiens	155-161	
20368952-4	2007	It was found that resveratrol inhibited the TNF-alpha-activated endothelial expression of vascular cell adhesion molecule-1 in a dose-dependent manner.	Resveratrol	18-29	vascular cell adhesion molecule 1	Homo sapiens	90-123	
9846848-4	1998	DESIGN: Because resveratrol is a tyrosine kinase inhibitor like other members of the tyrphostin family, we hypothesized that it has the ability to modify intracellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) expression by stimulated endothelial cells.	Resveratrol	16-27	vascular cell adhesion molecule 1	Homo sapiens	236-242	
9846848-6	1998	RESULTS: We showed that resveratrol, at concentrations as low as 1 micromol/L and 100 nmol/L, significantly inhibited ICAM-1 and VCAM-1 expression by tumor necrosis factor alpha (TNF-alpha)-stimulated human umbilical vein endothelial cells and lipopolysaccharide-stimulated human saphenous vein endothelial cells (HSVEC), respectively.	Resveratrol	24-35	vascular cell adhesion molecule 1	Homo sapiens	129-135