PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16624901-4	2006	In this study we analyzed the gene expression profile on a genomewide scale and found that deletion of either tsc1(+) or tsc2(+) affects gene induction upon nitrogen starvation.	Nitrogen	157-165	TSC complex subunit 1	Homo sapiens	110-114	
34085593-5	2021	Conversely, absence of a nitrogen source suppresses TORC1 in a manner dependent on GATOR1 as well as the Tsc1-Tsc2 complex, whose mammalian equivalent functions as a growth-factor sensitive TORC1 inhibitor.	Nitrogen	25-33	TSC complex subunit 1	Homo sapiens	105-109	
17179073-10	2007	Additionally, either Tsc1/2 or Tor1 are required for growth on a poor nitrogen source such as proline.	Nitrogen	70-78	TSC complex subunit 1	Homo sapiens	21-25	
17179073-11	2007	Mutants lacking Tsc1 or Tsc2 are highly sensitive to rapamycin under poor nitrogen conditions, suggesting that the function of Tor1 under such conditions is sensitive to rapamycin.	Nitrogen	74-82	TSC complex subunit 1	Homo sapiens	16-20	
34972198-5	2021	Loss of TSC1 and TSC2, that are negative regulators of TOR complex 1 (TORC1) in mammalian cells, resulted in altered nitrogen source-dependent growth of T. atroviride, reduced mycoparasitic overgrowth and, in the case of Deltatsc1, a diminished production of numerous secondary metabolites.	Nitrogen	117-125	TSC complex subunit 1	Homo sapiens	8-12	
25639242-4	2015	Nitrogen-stress-induced TORC1 inhibition differs from amino-acid-dependent control of TORC1 and requires the Ssp2 (AMPKalpha) kinase, the Tsc1/2 complex, and Rhb1 GTPase.	Nitrogen	0-8	TSC complex subunit 1	Homo sapiens	138-144