PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 26429394-5 2016 METHODS: In the current study we investigated the potential for AKR1C3 to regulate the availability of prostaglandin-derived ligands for PPARg mainly, prostaglandin J2 (PGJ2). 9-deoxy-delta-9-prostaglandin D2 151-167 aldo-keto reductase family 1 member C3 Homo sapiens 64-70 30012349-4 2019 By acting as an 11-ketoprostaglandin reductase, AKR1C3 generates 11beta-PGF2alpha to activate the FP receptor and deprives peroxisome proliferator activator receptorgamma of its putative PGJ2 ligands. 9-deoxy-delta-9-prostaglandin D2 187-191 aldo-keto reductase family 1 member C3 Homo sapiens 48-54 26429394-5 2016 METHODS: In the current study we investigated the potential for AKR1C3 to regulate the availability of prostaglandin-derived ligands for PPARg mainly, prostaglandin J2 (PGJ2). 9-deoxy-delta-9-prostaglandin D2 169-173 aldo-keto reductase family 1 member C3 Homo sapiens 64-70 20036328-8 2010 MCF-7-AKR1C3 cells also reduced PGD(2), limiting its dehydration to form PGJ(2) products. 9-deoxy-delta-9-prostaglandin D2 73-79 aldo-keto reductase family 1 member C3 Homo sapiens 6-12 24287039-7 2013 PPARgamma agonist (PGJ2) and antagonist (T0070907) enhanced PGFS mRNA abundance in the endometrium on days 10-12 and 14-16 of pregnancy, respectively. 9-deoxy-delta-9-prostaglandin D2 19-23 aldo-keto reductase family 1 member C3 Homo sapiens 60-64