PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18946034-1	2008	Human DNA polymerase eta (pol eta) can replicate across UV-induced pyrimidine dimers, and defects in the gene encoding pol eta result in a syndrome called xeroderma pigmentosum variant (XP-V).	pyrimidine	67-77	DNA polymerase eta	Homo sapiens	6-24	
25766642-2	2015	XP-V is caused by a deficiency in DNA polymerase eta (Poleta) that plays a pivotal role in translesion synthesis by bypassing UV-induced pyrimidine dimers.	pyrimidine	137-147	DNA polymerase eta	Homo sapiens	0-4	
25766642-2	2015	XP-V is caused by a deficiency in DNA polymerase eta (Poleta) that plays a pivotal role in translesion synthesis by bypassing UV-induced pyrimidine dimers.	pyrimidine	137-147	DNA polymerase eta	Homo sapiens	34-52	
20577208-1	2010	The variant form of the human syndrome xeroderma pigmentosum (XPV) is caused by a deficiency in DNA polymerase eta (Poleta), a DNA polymerase that enables replication through ultraviolet-induced pyrimidine dimers.	pyrimidine	195-205	DNA polymerase eta	Homo sapiens	96-114	
18946034-1	2008	Human DNA polymerase eta (pol eta) can replicate across UV-induced pyrimidine dimers, and defects in the gene encoding pol eta result in a syndrome called xeroderma pigmentosum variant (XP-V).	pyrimidine	67-77	DNA polymerase eta	Homo sapiens	186-190	
12082017-1	2002	Lack of DNA polymerase eta and the attendant defect in bypass replication of pyrimidine dimers induced in DNA by ultraviolet light (UV) underlie the enhanced mutagenesis and carcinogenesis observed in xeroderma pigmentosum variant (XP-V).	pyrimidine	77-87	DNA polymerase eta	Homo sapiens	232-236	
14643431-8	2003	This effect was reversed by expression of DNA polymerase eta, indicating that translesion synthesis of UVC-induced pyrimidine dimers by DNA pol eta protects human fibroblasts against UVC genotoxic effects even when other DNA repair functions are compromised by caffeine.	pyrimidine	115-125	DNA polymerase eta	Homo sapiens	42-60	
31053851-2	2019	One process is translesion synthesis (TLS) by DNA polymerases (Pol) delta, eta and zeta, which creates C>T transitions at pyrimidine dimers by incorporating two dAMPs opposite of the dimers.	pyrimidine	122-132	DNA polymerase eta	Homo sapiens	46-87