PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33837631-9	2021	These findings are consistent with the idea that CCCP-induced oxidative stress leading to eIF2alpha phosphorylation and ATF4 expression, which is known to stimulate genes involved in autophagy, play a pro-survival role together with AKT activation and regulate mTOR-mediated eIF4E-BP1 phosphorylation.	Carbonyl Cyanide m-Chlorophenyl Hydrazone	49-53	activating transcription factor 4	Homo sapiens	120-124	
33837631-1	2021	Mitochondrial dysfunction mediated by CCCP (carbonyl cyanide m-chlorophenyl hydrazone), an inhibitor of mitochondrial oxidative phosphorylation, evokes the integrated stress response (ISR), which is analyzed here by eIF2alpha phosphorylation and expression profiles of ATF4 and CHOP proteins.	Carbonyl Cyanide m-Chlorophenyl Hydrazone	38-42	activating transcription factor 4	Homo sapiens	269-273	
33837631-1	2021	Mitochondrial dysfunction mediated by CCCP (carbonyl cyanide m-chlorophenyl hydrazone), an inhibitor of mitochondrial oxidative phosphorylation, evokes the integrated stress response (ISR), which is analyzed here by eIF2alpha phosphorylation and expression profiles of ATF4 and CHOP proteins.	Carbonyl Cyanide m-Chlorophenyl Hydrazone	44-85	activating transcription factor 4	Homo sapiens	269-273	
33837631-6	2021	ISRIB, an inhibitor of the ISR pathway, mitigates CCCP-induced expression of ATF4 and CHOP, activation of AKT, and autophagy, similar to NAC.	Carbonyl Cyanide m-Chlorophenyl Hydrazone	50-54	activating transcription factor 4	Homo sapiens	77-81	
31116686-6	2019	Short-term menadione, antimycin A, or CCCP cell treatment led to the inhibition of protein synthesis, accompanied by a decrease in mTOR kinase activity, an increase in the phosphorylation of eIF2alpha (Ser51), and an increase in the level of ATF4 transcription factor.	Carbonyl Cyanide m-Chlorophenyl Hydrazone	38-42	activating transcription factor 4	Homo sapiens	242-246