PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18616671-7	2008	CONCLUSIONS: In both ethanol intoxication and thiamin-deficient glucose metabolism, BCSFB impairment exposes the CSF and hence the brain extracellular fluid to neuroactive substances from the blood.	Thiamine	46-53	colony stimulating factor 2	Rattus norvegicus	85-88	
7674851-0	1995	MRI demonstration of impairment of the blood-CSF barrier by glucose administration to the thiamin-deficient rat brain.	Thiamine	90-97	colony stimulating factor 2	Rattus norvegicus	45-48	
7674851-2	1995	The period of the onset of this blood-CSF or blood-brain barrier dysfunction coincides with our previous observations of accumulation of glutamate or glutamate derivatives following an equivalent glucose load under identical conditions of thiamin deficiency, consistent with a relationship between these two observations.	Thiamine	239-246	colony stimulating factor 2	Rattus norvegicus	38-41