PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11121595-6	2000	ET-1 reversed the reduction of CD44 expression by phosphoramidon.	phosphoramidon	50-64	endothelin 1	Mus musculus	0-4	
18758506-5	2008	In our previous work, we have shown that blockade of ET-1 synthesis through the use of the metalloproteinase inhibitor phosphoramidon results in control of preterm labor.	phosphoramidon	119-133	endothelin 1	Mus musculus	53-57	
11121595-10	2000	ET-1 reversed the suppression of oHA-induced proliferation by phosphoramidon.	phosphoramidon	62-76	endothelin 1	Mus musculus	0-4	
7781681-10	1995	Preincubation with phosphoramidon (100 microM) reduced responses to big-endothelin-1, but not endothelin-1.	phosphoramidon	19-33	endothelin 1	Mus musculus	72-84	
1826753-7	1991	The effect of big-ET-1[1-38], but not ET-1, was blocked by pretreatment with the enzyme inhibitor phosphoramidon (10 mg/kg, s.c., 30 min prior), implying that the big-ET-1[1-38] must first be enzymatically cleaved, presumably to ET-1, in order to elicit the abdominal constriction response.	phosphoramidon	98-112	endothelin 1	Mus musculus	18-22	
1579051-0	1992	Big endothelin-1-induced sudden death is inhibited by phosphoramidon in mice.	phosphoramidon	54-68	endothelin 1	Mus musculus	4-16	
1579051-4	1992	A metalloproteinase inhibitor, phosphoramidon, although failing to inhibit sudden death induced by ET-1, suppressed bET-1-induced lethality and elevation of plasma IR-ET-1 probably due to an inhibition of the enzymatic conversion of bET-1 to ET-1.	phosphoramidon	31-45	endothelin 1	Mus musculus	117-121	
1579051-4	1992	A metalloproteinase inhibitor, phosphoramidon, although failing to inhibit sudden death induced by ET-1, suppressed bET-1-induced lethality and elevation of plasma IR-ET-1 probably due to an inhibition of the enzymatic conversion of bET-1 to ET-1.	phosphoramidon	31-45	endothelin 1	Mus musculus	117-121