PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2775233-3	1989	Procaine and tetracaine inhibited acetylcholinesterase activity competitively at concentrations at which they did not perturb the physical state of the membrane lipid environment, as assessed by steady-state fluorescence polarization, whereas lidocaine and imipramine displayed mixed inhibition kinetics at concentrations at which they induced an enhancement of membrane fluidity.	Tetracaine	13-23	acetylcholinesterase (Cartwright blood group)	Homo sapiens	34-54	
11888725-2	2002	Addition of acetylcholine iodide or the competitive inhibitor tetracaine to the immobilized TPPS(1)-AChE complex results in a decrease in absorbance intensity at 446 nm due to displacement of the porphyrin from the active site.	Tetracaine	62-72	acetylcholinesterase (Cartwright blood group)	Homo sapiens	100-104	
3607051-1	1987	Release of acetylcholinesterase-containing vesicles from human erythrocyte membranes induced by dimyristoylphosphatidylcholine (DMPC) was inhibited by exposure of red cells to cationic amphiphilic drugs like tetracaine, chlorpromazine and primaquine which all are known to induce stomatocyte formation.	Tetracaine	208-218	acetylcholinesterase (Cartwright blood group)	Homo sapiens	11-31