PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18773427-3	2008	We report here that subconfluent PASMC express predominantly S1P2 and S1P3 receptors and we show that S1P1 receptor mRNA levels are significantly up-regulated following basic fibroblast growth factor (bFGF) treatment.	pasmc	33-38	fibroblast growth factor 2	Homo sapiens	201-205	
10516398-5	1999	Hypoxia (1% oxygen) and hypoxia-conditioned media increased PASMC numbers, and this mitogenic effect was abolished by anti-bFGF, but not by anti-PDGF or anti-VEGF.	pasmc	60-65	fibroblast growth factor 2	Homo sapiens	123-127	
17942638-9	2008	Furthermore, FGF-2 increased HIF-1alpha protein levels and consensus HRE promoter activity in PASMC via antioxidant-sensitive mechanisms.	pasmc	94-99	fibroblast growth factor 2	Homo sapiens	13-18	
17942638-10	2008	Thus we conclude that FGF-2 can stimulate its own expression in PASMC via NADPH oxidase-mediated activation of ROS-sensitive transcription factors, including HIF-1alpha.	pasmc	64-69	fibroblast growth factor 2	Homo sapiens	22-27	
10516398-7	1999	This suggests that PASMC-derived bFGF and VEGF may participate in hypoxic neonatal pulmonary vascular remodeling.	pasmc	19-24	fibroblast growth factor 2	Homo sapiens	33-37	
10397725-7	1999	By contrast, treatment of PASMC with serum and basic fibroblast growth factor (bFGF)/heparin markedly upregulated the expression of TFPI activity and antigen.	pasmc	26-31	fibroblast growth factor 2	Homo sapiens	47-77	
10397725-7	1999	By contrast, treatment of PASMC with serum and basic fibroblast growth factor (bFGF)/heparin markedly upregulated the expression of TFPI activity and antigen.	pasmc	26-31	fibroblast growth factor 2	Homo sapiens	79-83	
10397725-11	1999	Cycloheximide, but not actinomycin-D, treatment inhibited the serum and bFGF/heparin-induced increase in TFPI activity in PASMC.	pasmc	122-127	fibroblast growth factor 2	Homo sapiens	72-76