PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 18259028-2 2008 This study tested the hypothesis that 17beta-estradiol enhances the effect of angiotensin-converting enzyme inhibition on t-PA release in young postmenopausal women. Estradiol 38-54 angiotensin I converting enzyme Homo sapiens 78-107 10886123-9 2000 CONCLUSIONS: Acute administration of oestradiol caused vasodilation in males, the effect was not due to alterations in vascular ACE activity or to altered sensitivity to angiotensin II. Estradiol 37-47 angiotensin I converting enzyme Homo sapiens 128-131 10411297-6 1999 17Beta-oestradiol and oestrone significantly increased cellular ACE levels while 17beta,delta(8,9)-dehydroestradiol decreased cellular ACE. Estradiol 0-17 angiotensin I converting enzyme Homo sapiens 64-67 26679819-1 2015 Many studies identified new components of the renin-angiotensin system (RAS), such as Angiotensin-(1-7) [Ang-(1-7)] and Angiotensin-converting enzyme type 2 (ACE2), in mammalian ovaries.We previously showed Angiotensin-Converting Enzyme (ACE) inhibition, which increases the level of Ang-(1-7), stimulated ovarian estradiol output in ewe after estrous synchronization. Estradiol 314-323 angiotensin I converting enzyme Homo sapiens 158-161