PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18259028-2	2008	This study tested the hypothesis that 17beta-estradiol enhances the effect of angiotensin-converting enzyme inhibition on t-PA release in young postmenopausal women.	Estradiol	38-54	angiotensin I converting enzyme	Homo sapiens	78-107	
10886123-9	2000	CONCLUSIONS: Acute administration of oestradiol caused vasodilation in males, the effect was not due to alterations in vascular ACE activity or to altered sensitivity to angiotensin II.	Estradiol	37-47	angiotensin I converting enzyme	Homo sapiens	128-131	
10411297-6	1999	17Beta-oestradiol and oestrone significantly increased cellular ACE levels while 17beta,delta(8,9)-dehydroestradiol decreased cellular ACE.	Estradiol	0-17	angiotensin I converting enzyme	Homo sapiens	64-67	
26679819-1	2015	Many studies identified new components of the renin-angiotensin system (RAS), such as Angiotensin-(1-7) [Ang-(1-7)] and Angiotensin-converting enzyme type 2 (ACE2), in mammalian ovaries.We previously showed Angiotensin-Converting Enzyme (ACE) inhibition, which increases the level of Ang-(1-7), stimulated ovarian estradiol output in ewe after estrous synchronization.	Estradiol	314-323	angiotensin I converting enzyme	Homo sapiens	158-161