PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2431072-7	1986	These data suggest that the proteinaceous plasma-derived cofactor acts in a fashion similar to TPA and that this as yet unexplained mechanism of TPA action is important to the full expression of epibolin and to the early phase of epidermal cell spreading.	Tetradecanoylphorbol Acetate	145-148	vitronectin	Homo sapiens	195-203	
1696147-2	1990	The cells readily adhered on fibronectin, but TPA was needed for adherence on vitronectin and for the spreading of the cells on both substrata.	Tetradecanoylphorbol Acetate	46-49	vitronectin	Homo sapiens	78-89	
1696147-10	1990	Thus, the results suggest that TPA would activate several integrin receptors in HEL cells and also stimulate the secretion of thrombospondin, which might be used as an adhesion ligand for the integrin vitronectin receptor alpha v/beta 3 complex.	Tetradecanoylphorbol Acetate	31-34	vitronectin	Homo sapiens	201-212	
27267660-4	2016	Directly activating protein kinase Cs with phorbol myristate acetate stimulated microglial phagocytosis, and induced neuronal loss mediated by MFG-E8/vitronectin receptor pathway of microglial phagocytosis.	Tetradecanoylphorbol Acetate	43-68	vitronectin	Homo sapiens	150-161	
8616873-1	1996	Both TPA and A23187 dramatically enhanced MDA-MB-435 cell adhesion to type IV collagen (collagen IV), vitronectin, and, to some extent, fibronectin and laminin.	Tetradecanoylphorbol Acetate	5-8	vitronectin	Homo sapiens	102-113	
8616873-7	1996	Calphostin C, a PKC inhibitor, blocked the stimulation of adhesion by A23187, exogenous AA, and TPA to both collagen IV and vitronectin.	Tetradecanoylphorbol Acetate	96-99	vitronectin	Homo sapiens	124-135	
7543105-10	1995	The addition of the phorbol ester phorbol 12-myristate 13-acetate (PMA), a protein kinase C activator, to monolayers of human skin fibroblasts, increased vitronectin degradation.	Tetradecanoylphorbol Acetate	34-65	vitronectin	Homo sapiens	154-165	
7543105-10	1995	The addition of the phorbol ester phorbol 12-myristate 13-acetate (PMA), a protein kinase C activator, to monolayers of human skin fibroblasts, increased vitronectin degradation.	Tetradecanoylphorbol Acetate	67-70	vitronectin	Homo sapiens	154-165	
10205165-4	1999	Treatment with phorbol 12-myristate 13-acetate (PMA) allows alphavbeta3 to locate to the focal adhesion contacts and the cells to spread on vitronectin in the presence of CKI peptides.	Tetradecanoylphorbol Acetate	15-46	vitronectin	Homo sapiens	140-151	
10205165-4	1999	Treatment with phorbol 12-myristate 13-acetate (PMA) allows alphavbeta3 to locate to the focal adhesion contacts and the cells to spread on vitronectin in the presence of CKI peptides.	Tetradecanoylphorbol Acetate	48-51	vitronectin	Homo sapiens	140-151	
9066008-4	1997	Following stimulation with phorbolester (PMA), a 3-6 fold higher expression of uPA receptor over a period of up to 5 days could be observed by fluorescent activated cell-sorting as well as by direct ligand-binding of amino-terminal fragment of uPA or vitronectin.	Tetradecanoylphorbol Acetate	41-44	vitronectin	Homo sapiens	251-262	
8652819-3	1996	Using ultrastructural and confocal laser scanning microscopic (CLSM) image analysis, we observed that treatment of Dami cells, a human megakaryocytic cell line, with phorbol myristate acetate (PMA) induces the accumulation of PAI-1 and Vn in intracellular storage vacuoles that contain other alpha-granule proteins such as von Willebrand factor.	Tetradecanoylphorbol Acetate	166-191	vitronectin	Homo sapiens	236-238	
8652819-3	1996	Using ultrastructural and confocal laser scanning microscopic (CLSM) image analysis, we observed that treatment of Dami cells, a human megakaryocytic cell line, with phorbol myristate acetate (PMA) induces the accumulation of PAI-1 and Vn in intracellular storage vacuoles that contain other alpha-granule proteins such as von Willebrand factor.	Tetradecanoylphorbol Acetate	193-196	vitronectin	Homo sapiens	236-238