PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11404234-4	2001	Treatment with phorbol ester [12-O-tetradecanoyl-phorbol-13-acetate (TPA) 100 nM], an activator of protein kinase C, significantly enhanced 1,25(OH)2D3-induced OPN mRNA and transcription but had no effect on VDR or on 24(OH)ase mRNA or transcription.	Tetradecanoylphorbol Acetate	69-72	vitamin D receptor	Rattus norvegicus	208-211	
11404234-5	2001	Studies using OPN promoter constructs indicate that TPA-enhanced OPN transcription is mediated by an effect on the OPN promoter separate from an effect on VDR.	Tetradecanoylphorbol Acetate	52-55	vitamin D receptor	Rattus norvegicus	155-158	
8089198-5	1994	At 3 h after the treatment of the cells with H-7 and TPA, vitamin D receptor (VDR) contents estimated by 3H-1,25(OH)2D3 binding capacity were 72.4 and 63.2% of vehicle-treated cells without significant changes of binding affinities, suggesting that the effect of H-7 and TPA was not the result of changes in VDR content or its binding affinity.	Tetradecanoylphorbol Acetate	53-56	vitamin D receptor	Rattus norvegicus	58-76	
8089198-5	1994	At 3 h after the treatment of the cells with H-7 and TPA, vitamin D receptor (VDR) contents estimated by 3H-1,25(OH)2D3 binding capacity were 72.4 and 63.2% of vehicle-treated cells without significant changes of binding affinities, suggesting that the effect of H-7 and TPA was not the result of changes in VDR content or its binding affinity.	Tetradecanoylphorbol Acetate	53-56	vitamin D receptor	Rattus norvegicus	78-81	
8089198-5	1994	At 3 h after the treatment of the cells with H-7 and TPA, vitamin D receptor (VDR) contents estimated by 3H-1,25(OH)2D3 binding capacity were 72.4 and 63.2% of vehicle-treated cells without significant changes of binding affinities, suggesting that the effect of H-7 and TPA was not the result of changes in VDR content or its binding affinity.	Tetradecanoylphorbol Acetate	53-56	vitamin D receptor	Rattus norvegicus	308-311	
8089198-5	1994	At 3 h after the treatment of the cells with H-7 and TPA, vitamin D receptor (VDR) contents estimated by 3H-1,25(OH)2D3 binding capacity were 72.4 and 63.2% of vehicle-treated cells without significant changes of binding affinities, suggesting that the effect of H-7 and TPA was not the result of changes in VDR content or its binding affinity.	Tetradecanoylphorbol Acetate	271-274	vitamin D receptor	Rattus norvegicus	78-81	
9397948-2	1994	Activation of PKC with phorbol 12-myristate 13-acetate (PMA) resulted in a time- and dose-dependent increase in VDR expression in ROS cells.	Tetradecanoylphorbol Acetate	23-54	vitamin D receptor	Rattus norvegicus	112-115	
9397948-2	1994	Activation of PKC with phorbol 12-myristate 13-acetate (PMA) resulted in a time- and dose-dependent increase in VDR expression in ROS cells.	Tetradecanoylphorbol Acetate	56-59	vitamin D receptor	Rattus norvegicus	112-115	
9397948-8	1994	PMA treatment alone resulted in a 50% increase in VDR protein and a marginal 20% increase in VDR mRNA.	Tetradecanoylphorbol Acetate	0-3	vitamin D receptor	Rattus norvegicus	50-53	
9397948-8	1994	PMA treatment alone resulted in a 50% increase in VDR protein and a marginal 20% increase in VDR mRNA.	Tetradecanoylphorbol Acetate	0-3	vitamin D receptor	Rattus norvegicus	93-96	
1312452-8	1992	Coincubation for 4 h with PMA caused a decrease in PTH- and forskolin-induced up-regulation of VDR.	Tetradecanoylphorbol Acetate	26-29	vitamin D receptor	Rattus norvegicus	95-98	
20006981-5	2010	The PKCalpha activator, phorbol-12-myristate-13-acetate (PMA) induced the expression of VDR in the rat liver, and the induction of VDR by 1,25(OH)(2)D(3) and CDCA was inhibited by the PKCalpha inhibitor, bisindolyl maleimide I (Bis I).	Tetradecanoylphorbol Acetate	24-55	vitamin D receptor	Rattus norvegicus	88-91	
20006981-5	2010	The PKCalpha activator, phorbol-12-myristate-13-acetate (PMA) induced the expression of VDR in the rat liver, and the induction of VDR by 1,25(OH)(2)D(3) and CDCA was inhibited by the PKCalpha inhibitor, bisindolyl maleimide I (Bis I).	Tetradecanoylphorbol Acetate	24-55	vitamin D receptor	Rattus norvegicus	131-134	
20006981-5	2010	The PKCalpha activator, phorbol-12-myristate-13-acetate (PMA) induced the expression of VDR in the rat liver, and the induction of VDR by 1,25(OH)(2)D(3) and CDCA was inhibited by the PKCalpha inhibitor, bisindolyl maleimide I (Bis I).	Tetradecanoylphorbol Acetate	57-60	vitamin D receptor	Rattus norvegicus	88-91	
20006981-5	2010	The PKCalpha activator, phorbol-12-myristate-13-acetate (PMA) induced the expression of VDR in the rat liver, and the induction of VDR by 1,25(OH)(2)D(3) and CDCA was inhibited by the PKCalpha inhibitor, bisindolyl maleimide I (Bis I).	Tetradecanoylphorbol Acetate	57-60	vitamin D receptor	Rattus norvegicus	131-134