PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16670154-1	2006	There is evidence that increasing carbohydrate (CHO) availability during exercise by raising preexercise muscle glycogen levels attenuates the activation of AMPKalpha2 during exercise in humans.	CAV protocol	48-51	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	157-167	
24750267-3	2014	HYPOTHESIS/OBJECTIVES: To characterize tissue AMPK regulation in ponies subjected to a dietary carbohydrate (CHO) challenge.	CAV protocol	109-112	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	46-50	
24750267-7	2014	A high-CHO diet resulted in significantly decreased laminar [P-AMPK] in lean ponies (P = .03), but no changes in skeletal muscle (lean, P = .33; obese, P = .43) or liver (lean, P = .84; obese, P = .13) [P-AMPK].	CAV protocol	7-10	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	63-67	
24750267-8	2014	An inverse correlation existed between [blood glucose] and laminar [P-AMPK] in obese ponies on a high-CHO diet.	CAV protocol	102-105	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	70-74	
16670154-3	2006	We examined the effect of CHO ingestion on skeletal muscle AMPK signaling during exercise in nine active male subjects who completed two 120-min bouts of cycling exercise at 65 +/- 1% V(O2 peak).	CAV protocol	26-29	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	59-63