PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25769240-9	2015	EM900, CAM, or erythromycin significantly inhibited MUC5AC secretion induced by tumor necrosis factor-alpha from NCI-H292 cells.	Erythromycin	15-27	mucin 5AC, oligomeric mucus/gel-forming	Homo sapiens	52-58	
18339018-0	2008	Erythromycin attenuates MUC5AC synthesis and secretion in cultured human tracheal cells infected with RV14.	Erythromycin	0-12	mucin 5AC, oligomeric mucus/gel-forming	Homo sapiens	24-30	
18339018-6	2008	RESULTS: Erythromycin blocked RV14-induced MUC5AC protein overproduction and hypersecretion, and also blocked RV14-induced p44/42 MAPK activation in the cells.	Erythromycin	9-21	mucin 5AC, oligomeric mucus/gel-forming	Homo sapiens	43-49	
18339018-7	2008	CONCLUSIONS: Erythromycin may attenuate RV14-induced MUC5AC overproduction and hypersecretion by blocking the p44/42 MAPK pathway or its upstream regulators.	Erythromycin	13-25	mucin 5AC, oligomeric mucus/gel-forming	Homo sapiens	53-59	
12829454-5	2003	CAM and erythromycin significantly inhibited spontaneous and tumor necrosis factor-alpha (20 ng/ml)-induced mucus secretion from NCI-H292 cells at 10-6 to 10-7 M and from human nasal epithelial cells at 10-4 to 10-5 M. MUC5AC messenger RNA expression was also significantly inhibited.	Erythromycin	8-20	mucin 5AC, oligomeric mucus/gel-forming	Homo sapiens	219-225