PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15731493-4	2005	METHODS AND RESULTS: We demonstrate that insulin-mediated tyrosine phosphorylation of IRS-1 and serine phosphorylation of Akt, eNOS, and NO production are significantly inhibited by treatment of bovine aortic endothelial cells with 100 micromol/L FFA composed of palmitic acid for 3 hours before stimulation with 100 nM insulin.	Palmitic Acid	263-276	insulin	Bos taurus	41-48	
2668914-1	1989	In order to improve the lipophilicity of peptides, bovine insulin was chosen for the chemical modification using palmitic acid.	Palmitic Acid	113-126	insulin	Bos taurus	58-65	
27638262-3	2016	Because palmitic acid can promote insulin resistance in monogastrics through sphingolipid ceramide-dependent mechanisms, palmitic acid (C16:0) feeding may enhance milk production by restoring homeorhetic responses.	Palmitic Acid	8-21	insulin	Bos taurus	34-41	
24011949-14	2013	The PA treatment increased plasma concentration of nonesterified FA (101 vs. 90.0 muEq/L) and cholecystokinin (19.7 vs. 17.6 pmol/L), and tended to increase plasma concentration of insulin (10.7 vs. 9.57 mu IU/mL).	Palmitic Acid	4-6	insulin	Bos taurus	181-188