PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34151811-4	2021	METHODS: 200 and 400muM PA-conjugated BSA were applied to SH-SY5Y and HMC3 cells for 24 h. For FcgammaR blockage experiment, both cells were exposed to FcgammaR blocker before receiving of 200 and 400muM of PA-conjugated BSA for 24 h. RESULTS: PA significantly increased AD-related proteins, including Abeta and BACE1, as well as increasing TNFalpha, IL-1beta, and IL-6 in SH-SY5Y and HMC3 cells.	Palmitic Acid	244-246	amyloid beta precursor protein	Homo sapiens	302-307	
28659580-2	2017	The aim of this study was to investigate the non-genomic mechanism of extra-cellular palmitic acid (PA) regulating beta-amyloid peptide (Abeta) production, which may provide a link between obesity and the occurrence of AD.	Palmitic Acid	85-98	amyloid beta precursor protein	Homo sapiens	115-135	
28659580-2	2017	The aim of this study was to investigate the non-genomic mechanism of extra-cellular palmitic acid (PA) regulating beta-amyloid peptide (Abeta) production, which may provide a link between obesity and the occurrence of AD.	Palmitic Acid	100-102	amyloid beta precursor protein	Homo sapiens	115-135	
20383947-3	2010	Here, we show that PA treatment significantly reduced the expression of insulin-degrading enzyme (IDE), an important protease responsible for the degradation of amyloid-beta (A beta) in neural cells, while incubation with DHA up-regulated IDE levels in primary hippocampal neurons.	Palmitic Acid	19-21	amyloid beta precursor protein	Homo sapiens	161-173	
20383947-3	2010	Here, we show that PA treatment significantly reduced the expression of insulin-degrading enzyme (IDE), an important protease responsible for the degradation of amyloid-beta (A beta) in neural cells, while incubation with DHA up-regulated IDE levels in primary hippocampal neurons.	Palmitic Acid	19-21	amyloid beta precursor protein	Homo sapiens	175-181