PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23375034-3	2013	RESULTS: We found that the mRNA and protein expressions of TNF-alpha, IL-6, iNOS, COX-2 and NF-kappaB were significantly up-regulated in D-galactosamine induced hepatotoxic rats and treatment with carvacrol significantly down-regulated the expressions of these genes showing the mechanism behind the anti-inflammatory activity of carvacrol.	Galactosamine	137-152	interleukin 6	Rattus norvegicus	70-74	
17081566-9	2007	Incorporating these cells into the BAL device and testing in a GalN-induced FHF rat model resulted in significant reductions in plasma IL-6 levels and significantly improved animal survival.	Galactosamine	63-67	interleukin 6	Rattus norvegicus	135-139	
17081566-10	2007	Incorporating the AdIL-1Ra transfected TTNT cells in the BAL device and testing in the GalN-induced FHF rat model resulted in significantly reduced plasma IL-6 levels, and a trend toward improved survival was seen.	Galactosamine	87-91	interleukin 6	Rattus norvegicus	155-159	
11023982-4	2000	Hyper-IL-6 reversed the state of hepatotoxicity and enhanced the survival rates of rats suffering from fulminant hepatic failure after D-galactosamine administration.	Galactosamine	135-150	interleukin 6	Rattus norvegicus	6-10	
25978596-5	2016	Furthermore, isofuranodiene treatment significantly inhibited GalN/LPS-induced mRNA expression of IL-1beta, IL-6 and inducible nitric oxide synthase in liver tissues.	Galactosamine	62-66	interleukin 6	Rattus norvegicus	108-112	
25439027-8	2014	Increases in serum tumor necrosis factor alpha, interleukin-6, and interleukin-10, which were observed in GalN/LPS-treated rats, were significantly reduced after using SFN.	Galactosamine	106-110	interleukin 6	Rattus norvegicus	48-61