PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1472981-2	1992	An injection of D-galactosamine (GalN) into mice together with a lipopolysaccharide (LPS or endotoxin), interleukin-1 (IL-1) or tumour necrosis factor (TNF), sensitized the mice and induced fulminant hepatitis with severe congestion resulting in rapid death.	Galactosamine	16-31	interleukin 1 complex	Mus musculus	104-117	
10498828-5	1999	Administration of uridine (a precursor of UTP) prior injection of either LPS itself or interleukin-1 (IL-1) reduces the lethality of GalN+LPS.	Galactosamine	133-137	interleukin 1 complex	Mus musculus	87-106	
8758269-4	1996	The serum total bilirubin (TBIL) and liver necrosis of mice increased more markedly by using of TNF alpha, IL-6 or IFN gamma separately with D-GAL (TBIL: 46.2 +/- 10.6 micromol/L, 44.6 +/- 12.9 micromol/L, 41.9 +/- 14.9 micromol/L), then by D-GAL alone (TBIL: 27 +/- 11 micromol/L) also the serum TBIL of mice and liver necrosis also increased after injection of IL-1, IL-6 with D-GAL and TNF alpha.	Galactosamine	141-146	interleukin 1 complex	Mus musculus	363-367	
7509141-3	1993	Inhibition of liver protein synthesis with D-galactosamine (GALN) completely inhibited the IL-1-induced synthesis of acute-phase proteins.	Galactosamine	43-58	interleukin 1 complex	Mus musculus	91-95	
7509141-3	1993	Inhibition of liver protein synthesis with D-galactosamine (GALN) completely inhibited the IL-1-induced synthesis of acute-phase proteins.	Galactosamine	60-64	interleukin 1 complex	Mus musculus	91-95	
7509141-4	1993	GALN pretreatment abolished the protective effect of IL-1 on survival completely (neutropenic mice infected with Pseudomonas aeruginosa) or partially (nonneutropenic mice infected with Klebsiella pneumoniae).	Galactosamine	0-4	interleukin 1 complex	Mus musculus	53-57	
7489995-5	1995	In addition, pharmacological doses of the nitric oxide (NO) donor sodium nitroprusside conferred complete protection against TNF alpha-induced liver injury in galactosamine-sensitized mice, suggesting a possible link between IL-1- and NO-induced protection.	Galactosamine	159-172	interleukin 1 complex	Mus musculus	225-229	
1472981-21	1992	These results suggest that suppression of the induction of ODC by GalN may be one cause of the sensitization to LPS, IL-1 or TNF, and that the induction of HDC, i.e. histamine formation, may not be involved in this sensitization.9.	Galactosamine	66-70	interleukin 1 complex	Mus musculus	117-121	
2400992-1	1990	Both recombinant tumor necrosis factor (rTNF) and recombinant interleukin 1 (rIL-1) are able to mediate vascular collapse and death in a previously described murine model, using galactosamine to enhance the toxicity of these cytokines.	Galactosamine	178-191	interleukin 1 complex	Mus musculus	62-75