PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29555820-6	2018	We observed that a loss of Aurora A activity directly affects SAC function, that Aurora A is essential for maintaining the checkpoint protein Mad2 on unattached kinetochores and that inhibition of Aurora A leads to loss of the SAC, even in the presence of nocodazole or Taxol.	Nocodazole	256-266	aurora kinase A	Homo sapiens	27-35	
19221504-5	2009	Inhibition of Aurora A using MLN8054, a selective small-molecule inhibitor of Aurora A, in paclitaxel- or nocodazole-treated cells induces cells to become multinucleated.	Nocodazole	106-116	aurora kinase A	Homo sapiens	14-22	
21590355-7	2011	Knockdown of AURKA caused delayed entry into mitosis after treatment with nocodazole, reduced chromosomal instability, and decreased expression of focal adhesion kinase (FAK), phosphorylated FAK, and matrix metalloproteinase-2 (MMP-2), key regulators in cell adhesion and invasion.	Nocodazole	74-84	aurora kinase A	Homo sapiens	13-18	
21739483-5	2012	Nocodazole treatment arrested mitotic cells with multiple centrosomal Aurora A signals in CIN- and MIN-type CRC cells, albeit to a lower extent in CaCo-2 cells.	Nocodazole	0-10	aurora kinase A	Homo sapiens	70-78	
17591831-6	2007	With regard to the predictive role of Aurora-A, it has been shown that its overexpression disrupts the spindle checkpoint activated by paclitaxel (Taxol) or nocodazole treatment, thus inducing the cells to become resistant to these drugs.	Nocodazole	157-167	aurora kinase A	Homo sapiens	38-46	
17591831-7	2007	The development therefore of small molecules with an Aurora-A inhibition function may make it possible to reduce or block the oncogenic activity of Aurora-A and in addition may improve the survival of oncological patients showing resistance to paclitaxel or nocodazole treatment.	Nocodazole	258-268	aurora kinase A	Homo sapiens	53-61	
17591831-7	2007	The development therefore of small molecules with an Aurora-A inhibition function may make it possible to reduce or block the oncogenic activity of Aurora-A and in addition may improve the survival of oncological patients showing resistance to paclitaxel or nocodazole treatment.	Nocodazole	258-268	aurora kinase A	Homo sapiens	148-156	
16083279-6	2005	Total and fractionated extracts from nocodazole-treated HeLa cells were used as a source of Aurora-A substrates.	Nocodazole	37-47	aurora kinase A	Homo sapiens	92-100