PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23199370-3	2014	Treatment with microtubule-disrupting agents nocodazole and vinblastine suppressed meiotic resumption in a dose-dependent manner in both arrested cumulus cell-enclosed oocytes (CEO) stimulated with follicle-stimulating hormone (FSH) and arrested denuded oocytes (DO) stimulated with the AMPK activator, 5-aminoimidazole-4-carboxamide-1-beta-4-ribofuranoside (AICAR).	Nocodazole	45-55	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	287-291	
23199370-9	2014	Treatment with nocodazole led to disruption of proper spindle pole localization of active AMPK, while paclitaxel induced excessive polymerization of spindle MT and formation of ectopic asters with accentuated AMPK colocalization.	Nocodazole	15-25	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	90-94	
23199370-9	2014	Treatment with nocodazole led to disruption of proper spindle pole localization of active AMPK, while paclitaxel induced excessive polymerization of spindle MT and formation of ectopic asters with accentuated AMPK colocalization.	Nocodazole	15-25	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	209-213