PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33460615-0	2021	Gliclazide alters macrophages polarization state in diabetic atherosclerosis in vitro via blocking AGE-RAGE/TLR4-reactive oxygen species-activated NF-kbeta nexus.	Gliclazide	0-10	advanced glycosylation end product-specific receptor	Mus musculus	103-107	
33460615-10	2021	Glz pretreatment also effectively abated the AGEs-induced RAGE (~2-fold decrease), and CD86 surface marker expressions (P < 0.001 at 100 muM) on macrophages by inhibiting the NF-kbeta activation in a concentration dependent manner (1-100 muM) (P < 0.001).	Gliclazide	0-3	advanced glycosylation end product-specific receptor	Mus musculus	58-62	
33460615-11	2021	In conclusion, our data suggests that Glz alleviates the diabetic atherosclerosis progression by ameliorating the AGEs-mediated M1 pro-inflammatory phenotype via blocking AGE-RAGE/TLR4-reactive oxygen species -activated NF-kbeta nexus in macrophages.	Gliclazide	38-41	advanced glycosylation end product-specific receptor	Mus musculus	175-179