PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	45-49	
18728938-0	2008	Genetic variants of organic cation transporter 2 (OCT2) significantly reduce metformin uptake in oocytes.	Metformin	77-86	solute carrier family 22 member 2	Sus scrofa	20-48	
18728938-0	2008	Genetic variants of organic cation transporter 2 (OCT2) significantly reduce metformin uptake in oocytes.	Metformin	77-86	solute carrier family 22 member 2	Sus scrofa	50-54	
18728938-7	2008	Metformin uptake was much greater in oocytes expressing OCT2-wild type (OCT2-WT) than OCT1-WT compared with uptake in water-injected oocytes.	Metformin	0-9	solute carrier family 22 member 2	Sus scrofa	56-60	
18728938-7	2008	Metformin uptake was much greater in oocytes expressing OCT2-wild type (OCT2-WT) than OCT1-WT compared with uptake in water-injected oocytes.	Metformin	0-9	solute carrier family 22 member 2	Sus scrofa	72-76	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	135-144	solute carrier family 22 member 2	Sus scrofa	57-62	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	135-144	solute carrier family 22 member 2	Sus scrofa	57-61	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	135-144	solute carrier family 22 member 2	Sus scrofa	110-114	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	135-144	solute carrier family 22 member 2	Sus scrofa	110-114	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	274-283	solute carrier family 22 member 2	Sus scrofa	57-62	
18728938-8	2008	Uptake was significantly decreased in oocytes expressing OCT2-T199I, -T201M, and -A270S compared with that in OCT2-WT, suggesting that metformin is a better substrate for OCT2 than for OCT1 and that the amino acid-substituted variants of OCT2 cause a functional decrease in metformin uptake.	Metformin	274-283	solute carrier family 22 member 2	Sus scrofa	57-61	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	113-122	solute carrier family 22 member 2	Sus scrofa	39-43	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	113-122	solute carrier family 22 member 2	Sus scrofa	45-50	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	113-122	solute carrier family 22 member 2	Sus scrofa	45-49	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	39-43	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	45-50	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	45-49	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	39-43	
18728938-10	2008	In conclusion, the genetic variants of OCT2 (OCT2-T199I, -T201M, and -A270S) decreased the transport activity of metformin and thus may contribute to the inter-individual variation in metformin disposition as OCT2 plays a pivotal role in renal excretion, the major disposition route of metformin.	Metformin	184-193	solute carrier family 22 member 2	Sus scrofa	45-50