PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32901466-9	2020	However, compared to the wildtype (WT) cells, exposure to either tBHP or ROT enhanced the production of ROS, ECAR, and the proton (H+) production rate (PPR) from glycolysis, as well as promoted apoptotic cell death in VDAC1-/- H9c2 cells.	tert-Butylhydroperoxide	65-69	voltage-dependent anion channel 1	Rattus norvegicus	218-223	
32901466-11	2020	Restoration of VDAC1 in VDAC1-/- H9c2 cells reinstated mitochondria-bound HKII and concomitantly decreased tBHP and ROT-induced ROS production and cell death.	tert-Butylhydroperoxide	107-111	voltage-dependent anion channel 1	Rattus norvegicus	15-20	
32901466-11	2020	Restoration of VDAC1 in VDAC1-/- H9c2 cells reinstated mitochondria-bound HKII and concomitantly decreased tBHP and ROT-induced ROS production and cell death.	tert-Butylhydroperoxide	107-111	voltage-dependent anion channel 1	Rattus norvegicus	24-29	
32901466-12	2020	Interestingly, mitochondrial respiration remained the same after tBHP treatment in VDAC1-/- and WT H9c2 cells.	tert-Butylhydroperoxide	65-69	voltage-dependent anion channel 1	Rattus norvegicus	83-88