PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27498916-9	2016	The expression of Mac-1 and L-selectin decreased by 51.0% (P< .01) and 30.9% (P= .02), respectively, following face mask inhalation of budesonide and by 39.8% (P= .01) and 17.4% (P= .17), respectively, following inhalation of fluticasone.	Budesonide	138-148	integrin subunit alpha M	Homo sapiens	18-23	
10837366-4	2000	Budesonide, hydrocortisone, and prednisolone, but not the sex steroids testosterone and progesterone, reduced CD11b and CD49d cell-surface expression to a similar extent.	Budesonide	0-10	integrin subunit alpha M	Homo sapiens	110-115	
11029322-4	2000	We measured the modulation of expression of clonal designator (CD)11b and L-selectin with flow cytometry after 4 h or 16 h of culture of eosinophils when budesonide or formoterol was applied either directly to the eosinophils while they were stimulated with FCM (direct method) or when each drug was applied to lung fibroblasts from which conditioned medium was then administered to eosinophils (indirect method).	Budesonide	154-164	integrin subunit alpha M	Homo sapiens	63-69	
11029322-5	2000	In the direct method, budesonide (10(-)(8) M) inhibited the modulation of CD11b (44 [25th to 75th percentiles: 26 to 66]% of control) and L-selectin (30 [-13 to 48]% of control) only after 16 h, and not after 4 h. Formoterol did not directly inhibit the modulation of eosinophil CD11b and L-selectin expression.	Budesonide	22-32	integrin subunit alpha M	Homo sapiens	74-79	
11029322-5	2000	In the direct method, budesonide (10(-)(8) M) inhibited the modulation of CD11b (44 [25th to 75th percentiles: 26 to 66]% of control) and L-selectin (30 [-13 to 48]% of control) only after 16 h, and not after 4 h. Formoterol did not directly inhibit the modulation of eosinophil CD11b and L-selectin expression.	Budesonide	22-32	integrin subunit alpha M	Homo sapiens	279-284	
11029322-6	2000	In the indirect method, both budesonide and formoterol inhibited lung fibroblast activation, resulting in diminished eosinophil activation after 4 h. Budesonide or formoterol at 10(-)(8) M inhibited upregulation of CD11b to 26 [15 to 40]% and 38 [23 to 46]%, respectively, and inhibited L-selectin shedding to 14 [-3 to 50]% and 27 [2 to 62]%, respectively, of control values.	Budesonide	29-39	integrin subunit alpha M	Homo sapiens	215-220	
11029322-6	2000	In the indirect method, both budesonide and formoterol inhibited lung fibroblast activation, resulting in diminished eosinophil activation after 4 h. Budesonide or formoterol at 10(-)(8) M inhibited upregulation of CD11b to 26 [15 to 40]% and 38 [23 to 46]%, respectively, and inhibited L-selectin shedding to 14 [-3 to 50]% and 27 [2 to 62]%, respectively, of control values.	Budesonide	150-160	integrin subunit alpha M	Homo sapiens	215-220	
10718988-6	2000	Preincubation of the cells with different concentrations of budesonide was also effective in down-regulating the C5a-induced ICAM-1 expression on HBECs and the ah-CD23 and GM-CSF-induced LFA-1 and Mac-1 expression on eosinophils.	Budesonide	60-70	integrin subunit alpha M	Homo sapiens	197-202	
30236840-8	2018	Further, we show that the budesonide NS enema treated mice had a significantly reduced number of inflammatory macrophages and IL-beta producing CD11b + cells in colon tissue compared to untreated controls or mice treated with the budesonide MS enema.	Budesonide	26-36	integrin subunit alpha M	Homo sapiens	144-149	
30236840-8	2018	Further, we show that the budesonide NS enema treated mice had a significantly reduced number of inflammatory macrophages and IL-beta producing CD11b + cells in colon tissue compared to untreated controls or mice treated with the budesonide MS enema.	Budesonide	230-240	integrin subunit alpha M	Homo sapiens	144-149