PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8603407-2	1996	The arrested WEHI cells (60-75% in G0-G1) showed increased sensitivity to TNF killing, measured as 48-h 3-(5-dimethylthiazol-2-yl)-2,5-diphenyl-tetrazolium bromide assays, and 15-h apoptosis by propidium iodide staining and flow cytometry analysis.	Propidium	194-210	tumor necrosis factor	Mus musculus	74-77	
32108953-6	2020	IL-1beta/TNF death was necrosis by trypan blue and propidium iodide positivity, absence of mitochondrial death pathway and caspase activation, and failure of a caspase inhibitor or necrostatin-1s to prevent death.	Propidium	51-67	tumor necrosis factor	Mus musculus	9-12	
10455154-6	1999	Here we report that, although L929 cells underwent DNA fragmentation after exposure to TNF, they retained the morphology of intact nuclei while gaining permeability to propidium iodide, features characteristic of necrosis rather than apoptosis.	Propidium	168-184	tumor necrosis factor	Mus musculus	87-90	
8747598-4	1995	The onset of cellular damage in TNFs cells, as indicated by propidium iodide uptake, was seen as early as 6 h after TNF-alpha treatment.	Propidium	60-76	tumor necrosis factor	Mus musculus	116-125	
8613675-3	1996	The method is based on the binding of propidium iodide (PI), a membrane-impermeant dye, to nucleic acids of WEHI 164 cells, whose plasma membrane permeable due to TNF-alpha-induced cell damage.	Propidium	38-54	tumor necrosis factor	Mus musculus	163-172	
8186366-1	1993	Early events in the cytotoxic response to tumour necrosis factor (TNF) of the murine fibrosarcoma cell lines L929 and WEHI164cl13 were assessed on a cell by cell basis using the fluorescent exclusion dye propidium iodide (PI) and analysis by flow cytometry.	Propidium	204-220	tumor necrosis factor	Mus musculus	66-69