PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31123185-1	2019	Serotonin (5-HT) is known to increase the rate of growth cone advance via cofilin-dependent increases in retrograde actin network flow and nonmuscle myosin II activity.	Serotonin	0-9	cofilin 1	Homo sapiens	74-81	
31123185-1	2019	Serotonin (5-HT) is known to increase the rate of growth cone advance via cofilin-dependent increases in retrograde actin network flow and nonmuscle myosin II activity.	Serotonin	11-15	cofilin 1	Homo sapiens	74-81	
31123185-3	2019	5-HT simultaneously induces cofilin-dependent decreases in actin network density and PKC-dependent increases in point contact density.	Serotonin	0-4	cofilin 1	Homo sapiens	28-35	
31123185-5	2019	Interestingly, when PKC activity was up-regulated, 5-HT treatments resulted in myosin II hyperactivation accompanied by catastrophic cofilin-dependent decreases in actin filament density, sudden decreases in traction force, and neurite retraction.	Serotonin	51-55	cofilin 1	Homo sapiens	133-140	
16247020-5	2005	Consistent with this structural change by cofilin overexpression, the synaptic strength was reduced, and furthermore, the long-term facilitation elicited by repeated pulses of 5-hydroxytryptamine was impaired in sensory-to-motor synapses.	Serotonin	176-195	cofilin 1	Homo sapiens	42-49	
32060357-3	2020	Here, we employ high-resolution time-lapse FRET imaging in neuroblastoma cells and neuronal dendrites to establish that activation of serotonin receptor 5-HT4 (5-HT4R) rapidly triggers spatially-restricted RhoA activity and G13-mediated phosphorylation of cofilin, thus locally boosting the filamentous actin fraction.	Serotonin	134-143	cofilin 1	Homo sapiens	256-263