PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33560400-8	2021	Notably, an antioxidant, N-acetyl-l-cysteine, rescued HSC and lymphoid progenitor cell depletion, indicating a causal impact of OXPHOS-mediated ROS generation upon Jmjd6 deletion.	Acetylcysteine	25-44	fucosyltransferase 1 (H blood group)	Homo sapiens	54-57	
32655795-4	2020	In addition, N-acetylcysteine (NAC) has been shown to effectively suppress HSC activation-dependent expression of alpha-smooth muscle actin in HSCs, suggesting that NAC could be a candidate for the clinical treatment of hepatic fibrosis.	Acetylcysteine	13-29	fucosyltransferase 1 (H blood group)	Homo sapiens	75-78	
32655795-4	2020	In addition, N-acetylcysteine (NAC) has been shown to effectively suppress HSC activation-dependent expression of alpha-smooth muscle actin in HSCs, suggesting that NAC could be a candidate for the clinical treatment of hepatic fibrosis.	Acetylcysteine	31-34	fucosyltransferase 1 (H blood group)	Homo sapiens	75-78	
32655795-4	2020	In addition, N-acetylcysteine (NAC) has been shown to effectively suppress HSC activation-dependent expression of alpha-smooth muscle actin in HSCs, suggesting that NAC could be a candidate for the clinical treatment of hepatic fibrosis.	Acetylcysteine	165-168	fucosyltransferase 1 (H blood group)	Homo sapiens	75-78	
32655795-7	2020	In addition, co-administration of NAC with everolimus further reduced the expression of fibrogenic genes and improved the characteristic of HSCs via blockage of HSC activation and up-regulation of fibrolytic gene.	Acetylcysteine	34-37	fucosyltransferase 1 (H blood group)	Homo sapiens	140-143	
31827678-10	2019	Lastly, we discuss the possibility of using small molecule antioxidants, such as N-acetyl cysteine, resveratrol, and curcumin, to augment HSC function and improve the therapeutic efficacy of BM transplantation.	Acetylcysteine	81-98	fucosyltransferase 1 (H blood group)	Homo sapiens	138-141