PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26303143-5	2015	CONCLUSION: This study reported for the first time that long-term elevated plasma concentration of NPY could induce cardiac dysfunction and cardiac hypertrophy and this phenomenon could, in part, be mediated by the Ca2+/CaM-dependent CaN pathway and p38 mitogen-activated protein kinase (MAPK) signal pathway in rats.	cafestol palmitate	220-223	neuropeptide Y	Rattus norvegicus	99-102	
31430504-3	2019	Eleven weeks of CAF (medium-term) increased energy intake, adiposity, leptinemia, and body weight, with an orexigenic response of the lateral hypothalamus, paraventricular and ventromedial nuclei, given by upregulation of Orexins, AgRP, and NPY opposed by an anorectic signal of the arcuate nucleus, which displayed a higher POMC expression.	cafestol palmitate	16-19	neuropeptide Y	Rattus norvegicus	241-244	
15582708-3	2005	This study tested the hypothesis that NPY induces cardiomyocyte hypertrophy through Ca2+/CaM-dependent calcineurin (CaN) pathway in cultured neonatal rat cardiomyocytes.	cafestol palmitate	89-92	neuropeptide Y	Rattus norvegicus	38-41	
15582708-9	2005	The result suggested that NPY could induce hypertrophy of cardiomyocytes via Ca2+/CaM-dependent CaN signal pathway.	cafestol palmitate	82-85	neuropeptide Y	Rattus norvegicus	26-29