PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30820155-7	2019	Our mitogen-activated protein kinase inhibitor analysis revealed that phosphorylation of c-Jun N-terminal kinase (JNK), but not extracellular signal-regulated kinase or p38, mediates UVA-induced AQP3 downregulation, and that MEL treatment significantly suppressed the UVA-induced phosphorylation of JNK.	uva	183-186	mitogen-activated protein kinase 8	Homo sapiens	89-112	
30820155-7	2019	Our mitogen-activated protein kinase inhibitor analysis revealed that phosphorylation of c-Jun N-terminal kinase (JNK), but not extracellular signal-regulated kinase or p38, mediates UVA-induced AQP3 downregulation, and that MEL treatment significantly suppressed the UVA-induced phosphorylation of JNK.	uva	183-186	mitogen-activated protein kinase 8	Homo sapiens	299-302	
30820155-7	2019	Our mitogen-activated protein kinase inhibitor analysis revealed that phosphorylation of c-Jun N-terminal kinase (JNK), but not extracellular signal-regulated kinase or p38, mediates UVA-induced AQP3 downregulation, and that MEL treatment significantly suppressed the UVA-induced phosphorylation of JNK.	uva	268-271	mitogen-activated protein kinase 8	Homo sapiens	89-112	
30820155-7	2019	Our mitogen-activated protein kinase inhibitor analysis revealed that phosphorylation of c-Jun N-terminal kinase (JNK), but not extracellular signal-regulated kinase or p38, mediates UVA-induced AQP3 downregulation, and that MEL treatment significantly suppressed the UVA-induced phosphorylation of JNK.	uva	268-271	mitogen-activated protein kinase 8	Homo sapiens	114-117	
30820155-7	2019	Our mitogen-activated protein kinase inhibitor analysis revealed that phosphorylation of c-Jun N-terminal kinase (JNK), but not extracellular signal-regulated kinase or p38, mediates UVA-induced AQP3 downregulation, and that MEL treatment significantly suppressed the UVA-induced phosphorylation of JNK.	uva	183-186	mitogen-activated protein kinase 8	Homo sapiens	114-117	
19119326-10	2008	EGCG also inhibited UVA-induced extracullar signal-regulated kinase (ERK) and c-jun-NH2 terminal kinase (JNK) activation in RPE cells while a higher concentration of EGCG had an inhibitory effect on UVA-induced p38 activation.	uva	20-23	mitogen-activated protein kinase 8	Homo sapiens	105-108	
23322578-9	2013	The ERK and JNK inhibitor, but not p38 MAPK inhibitors, significantly decreased the UVA-induced expression of uPA by pterygium fibroblasts.	uva	84-87	mitogen-activated protein kinase 8	Homo sapiens	12-15	
22443056-0	2012	[Protection of HSF1/HSP70 pathway on UVA-induced HaCaT cells apoptosis via inhibiting the activation of c-Jun N-terminal kinase].	uva	37-40	mitogen-activated protein kinase 8	Homo sapiens	104-127	
15018897-8	2004	Since MAPKs, specifically p38 and JNK, appear to play a major role in the expression of UVA-induced AP-1 and COX-2, pharmacological inhibitors may be of benefit in the chemoprevention of non-melanoma skin cancer.	uva	88-91	mitogen-activated protein kinase 8	Homo sapiens	34-37	
16613495-8	2006	In conclusion, we have observed that inhibition of UVA-induced JNK activity with the pharmacologic inhibitor SP600125 resulted in caspase-dependent apoptotic cell death in both the immortalized keratinocyte cell line HaCaT and primary keratinocytes.	uva	51-54	mitogen-activated protein kinase 8	Homo sapiens	63-66