PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30772783-11	2019	Mechanistically, we showed that TFA increased the expression of anti-apoptotic factor such as Bcl-2 and survivin, decreased the expression of pro-apoptotic factor such as Caspase 3, Bax and Bak and inhibited the activation of caspase 3 and 9.	Trifluoroacetic Acid	32-35	BCL2 associated X, apoptosis regulator	Rattus norvegicus	182-185	
28856926-7	2018	Moreover, TFA significantly increased the ratios of P-Akt/Akt and P-eNOS/eNOS in vascular endothelial cells under hypoxic conditions, but did not change the P-Erk/Erk or Bcl-2/Bax ratios.	Trifluoroacetic Acid	10-13	BCL2 associated X, apoptosis regulator	Rattus norvegicus	176-179	
23442644-6	2012	Furthermore, the prostatic expression of Blc-2 was inhibited, whereas that of Bax and p53 was activated by TFA and AHT.	Trifluoroacetic Acid	107-110	BCL2 associated X, apoptosis regulator	Rattus norvegicus	78-81