PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30223923-9	2018	Further studies revealed that TFA significantly inhibited iNOS and COX-2 protein levels, the phosphorylations of p38 and JNK in MAPKs pathway and IKKalpha/beta, IkappaBalpha and the expression of nuclear NF-kappaB p65 in NF-kappaB pathway in LPS-stimulated RAW 264.7 cells.	Trifluoroacetic Acid	30-33	mitogen-activated protein kinase 8	Mus musculus	121-124	
33091854-8	2021	Further study demonstrated that TFA repressed the phosphorylation, nuclear translocation and transcriptional activity of NFkappaB, and inhibited the activation of AKT and JNK signaling in BV-2 cells induced by LPS.	Trifluoroacetic Acid	32-35	mitogen-activated protein kinase 8	Mus musculus	171-174	
33091854-9	2021	The agonists of AKT and JNK, anisomycin and SC79, could partly abolish the inhibitory effect of TFA on the production of inflammatory mediators in BV-2 cells induced by LPS.	Trifluoroacetic Acid	96-99	mitogen-activated protein kinase 8	Mus musculus	24-27	
33091854-10	2021	CONCLUSIONS: Taken together, our results clarified that TFA inhibited microglia-mediated inflammation in EAE mice probably through deactivating JNK/AKT/NFkappaB signaling pathways.	Trifluoroacetic Acid	56-59	mitogen-activated protein kinase 8	Mus musculus	144-147