PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31504964-5	2019	Monocrotaline-induced HSOS in mice and rats was evidenced by the increased serum alanine/aspartate aminotransferase (ALT/AST) activities, the elevated hepatic metalloproteinase 9 (MMP9) expression, and results from liver histological evaluation and scanning electron microscope observation.	Monocrotaline	0-13	matrix metallopeptidase 9	Rattus norvegicus	180-184	
31922224-9	2020	The results demonstrated that FMN significantly alleviated the changes of hemodynamics and pulmonary vascular morphology, and decreased the MCT-induced upregulations of TGFbeta1, MMP2 and MMP9 expression levels.	Monocrotaline	140-143	matrix metallopeptidase 9	Rattus norvegicus	188-192	
12949732-4	2003	RESULTS: In the monocrotaline-induced rat model of sinusoidal obstruction syndrome, there was an early increase of matrix metalloproteinase-9 and a later, lower-magnitude increase of matrix metalloproteinase-2 in the liver.	Monocrotaline	16-29	matrix metallopeptidase 9	Rattus norvegicus	115-141	
12949732-5	2003	In vitro studies of sinusoidal endothelial cells, hepatocytes, stellate cells, and Kupffer cells showed that sinusoidal endothelial cells are the major source of both basal and monocrotaline-induced matrix metalloproteinase-9/matrix metalloproteinase-2 activity.	Monocrotaline	177-190	matrix metallopeptidase 9	Rattus norvegicus	199-252	
19057128-12	2008	MMP-2 and MMP-9 expressions and activities in right ventricles increased significantly in monocrotaline-injected rats and captopril inhibited them.	Monocrotaline	90-103	matrix metallopeptidase 9	Rattus norvegicus	10-15