PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11590204-6	2001	The tyrosine-phosphorylation patterns induced by MSU crystals and by the ligation of CD16 were inhibited by piceatannol, which, conversely, only slightly delayed but did not diminish the peak of tyrosine phosphorylation induced by cross-linking CD32 or by the addition of fMet-Leu-Phe.	N-Formylmethionine Leucyl-Phenylalanine	272-284	Fc gamma receptor IIIa	Homo sapiens	85-89	
8223854-3	1993	In granulocytes primed with 200 mM N-formyl-Met-Leu-Phe (FMLP), cross-linking of cell-bound anti-CD16, -CD24, -CD59 and -CD67 led to calcium fluxes and activation of the oxidative burst.	N-Formylmethionine Leucyl-Phenylalanine	35-55	Fc gamma receptor IIIa	Homo sapiens	97-101	
1979013-4	1990	When neutrophils were incubated in the presence of cycloheximide, they progressively lost their ability to generate reactive oxidants in response to fMet-Leu-Phe so that by 5 h incubation with this inhibitor they could only generate about 25% of the oxidative response stimulated in untreated cells, and the expression of CD16 and CD18 was grossly impaired.	N-Formylmethionine Leucyl-Phenylalanine	149-161	Fc gamma receptor IIIa	Homo sapiens	322-326