PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32446776-7	2020	Functional analyses results showed that the differential genes mainly related with posttranscriptional regulation of gene expression and protein localization to organelle and so on, which involved in AMPK, PI3K-Akt signaling pathway, and GABA-ergic synapse.	gamma-Aminobutyric Acid	238-242	thymoma viral proto-oncogene 1	Mus musculus	211-214	
30079374-8	2018	These results indicate that estradiol acts directly on GnRH neurons via the ERbeta/Akt/nNOS pathway at proestrus afternoon generating NO that retrogradely accelerates GABA and glutamate release from the presynaptic terminals contacting GnRH neurons.	gamma-Aminobutyric Acid	167-171	thymoma viral proto-oncogene 1	Mus musculus	83-86	
27615800-0	2016	Akting up in the GABA hypothesis of schizophrenia: Akt1 deficiency modulates GABAergic functions and hippocampus-dependent functions.	gamma-Aminobutyric Acid	17-21	thymoma viral proto-oncogene 1	Mus musculus	51-55	
21709230-5	2011	Unlike in adult brain or islet alpha-cells in which GABA exerts hyperpolarizing effects, in islet beta-cells, GABA produces membrane depolarization and Ca(2+) influx, leading to the activation of PI3-K/Akt-dependent growth and survival pathways.	gamma-Aminobutyric Acid	110-114	thymoma viral proto-oncogene 1	Mus musculus	202-205	
34296180-5	2021	In mice, we find that AKT1 is the most broadly expressed isoform, with expression in excitatory neurons and the sole detectable AKT isoform in gamma-aminobutyric acid ergic interneurons and microglia.	gamma-Aminobutyric Acid	143-166	thymoma viral proto-oncogene 1	Mus musculus	128-131	
22385968-2	2012	Here we show that regulation of dendritic development of newborn neurons by Disrupted-in-Schizophrenia 1 (DISC1) during adult hippocampal neurogenesis requires neurotransmitter GABA-induced, NKCC1-dependent depolarization through a convergence onto the AKT-mTOR pathway.	gamma-Aminobutyric Acid	177-181	thymoma viral proto-oncogene 1	Mus musculus	253-256	
35456004-4	2022	Studies have indicated the involvement of FMRP in a multitude of cellular pathways, and an absence of FMRP was shown to affect several neurotransmitter receptors, for example, the GABA receptor and intracellular signaling molecules such as Akt, ERK, mTOR, and GSK3.	gamma-Aminobutyric Acid	180-184	thymoma viral proto-oncogene 1	Mus musculus	240-243