PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27098371-0	2016	Altered development in GABA co-release shapes glycinergic synaptic currents in cultured spinal slices of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.	gamma-Aminobutyric Acid	23-27	superoxide dismutase 1, soluble	Mus musculus	109-113	
29400109-3	2019	Region-specific neurochemical alterations were detected in asymptomatic G93A-SOD1 mice, particularly in lactate (-19%) and glutamate (+8%) of brainstem, along with gamma-amino-butyric acid (-30%), N-acetyl-aspartate (-5%) and ascorbate (+51%) of motor cortex.	gamma-Aminobutyric Acid	164-188	superoxide dismutase 1, soluble	Mus musculus	77-81	
27098371-10	2016	We performed single cell electrophysiology, immunocytochemistry and confocal microscopy and suggest that GABA co-release may speed the decay of glycine responses altering both temporal precision and signal integration in SOD1(G93A) developing networks at the postsynaptic site.	gamma-Aminobutyric Acid	105-109	superoxide dismutase 1, soluble	Mus musculus	221-225	
27098371-11	2016	Our hypothesis is supported by the finding of an increased MN bursting activity in immature SOD1(G93A) spinal cords and by immunofluorescence microscopy detection of a longer persistence of GABA in SOD1(G93A) glycinergic terminals in cultured and ex vivo spinal slices.	gamma-Aminobutyric Acid	190-194	superoxide dismutase 1, soluble	Mus musculus	198-202	
16087196-0	2005	The cellular mRNA expression of GABA and glutamate receptors in spinal motor neurons of SOD1 mice.	gamma-Aminobutyric Acid	32-36	superoxide dismutase 1, soluble	Mus musculus	88-92	
15885796-6	2005	The GLY-evoked release of [3H]d-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]d-ASP release, but not that of [3H]GLY, were more pronounced in SOD1-G93A(+) than in SOD1(+) or SOD1(-)/G93A(-) mice.	gamma-Aminobutyric Acid	71-75	superoxide dismutase 1, soluble	Mus musculus	151-155	
15885796-7	2005	Furthermore, the excessive potentiation of [3H]d-ASP by GLY or GABA was already present in asymptomatic 30-40 day-old SOD1-G93A(+) mice.	gamma-Aminobutyric Acid	63-67	superoxide dismutase 1, soluble	Mus musculus	118-122	
15885796-8	2005	The releases of endogenous glutamate and GABA also were enhanced by GLY and the GLY-evoked release of endogenous glutamate, but not of endogenous GABA, was higher in SOD1-G93A(+) than in control animals.	gamma-Aminobutyric Acid	41-45	superoxide dismutase 1, soluble	Mus musculus	166-170	
15885796-6	2005	The GLY-evoked release of [3H]d-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]d-ASP release, but not that of [3H]GLY, were more pronounced in SOD1-G93A(+) than in SOD1(+) or SOD1(-)/G93A(-) mice.	gamma-Aminobutyric Acid	71-75	superoxide dismutase 1, soluble	Mus musculus	172-176	
15885796-6	2005	The GLY-evoked release of [3H]d-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]d-ASP release, but not that of [3H]GLY, were more pronounced in SOD1-G93A(+) than in SOD1(+) or SOD1(-)/G93A(-) mice.	gamma-Aminobutyric Acid	71-75	superoxide dismutase 1, soluble	Mus musculus	172-176	
15033338-3	2004	In SOD1(-)/G93A(-) or SOD1(+) mice glycine evoked [(3)H]d-ASP and [(3)H]GABA release, while GABA caused [3H]D-ASP, but not [3H]glycine, release.	gamma-Aminobutyric Acid	92-96	superoxide dismutase 1, soluble	Mus musculus	3-7	
15033338-4	2004	The glycine-evoked release of [3H]D-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]D-ASP release, but not that of [3H]glycine, were more pronounced in SOD1-G93A(+) than in SOD1(+) mice.	gamma-Aminobutyric Acid	75-79	superoxide dismutase 1, soluble	Mus musculus	159-163	
15033338-4	2004	The glycine-evoked release of [3H]D-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]D-ASP release, but not that of [3H]glycine, were more pronounced in SOD1-G93A(+) than in SOD1(+) mice.	gamma-Aminobutyric Acid	75-79	superoxide dismutase 1, soluble	Mus musculus	180-184