PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25505315-6	2014	Moreover, TRPV1-mediated depression occurs independently of GABA release, requires postsynaptic Ca(2+) rise and activation of calcineurin, and is likely due to clathrin-dependent internalization of GABA receptors.	gamma-Aminobutyric Acid	60-64	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	10-15	
21672630-5	2011	By means of whole-cell patch clamp experiments in corticostriatal brain slices, we found that the absence of TRPV1 channels exacerbated the defect of glutamate transmission occurring in the peak phase of EAE, and attenuated the alterations of GABA synapses in the chronic phase of EAE, thus paralleling the dual effects of TRPV1-KO on the motor deficits of EAE mice.	gamma-Aminobutyric Acid	243-247	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	109-114	
22074644-10	2012	The expression of GABA(A)gamma(2) was higher in the NAc, CPU, and cortex of TRPV1KO versus WT mice, whereas the expression of GABA(A)alpha(2) was lower in the FrA, CPU, NAc, and cortex in TRPV1KO mice than WT mice.	gamma-Aminobutyric Acid	18-22	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	76-81