PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32147148-10	2020	IMPLICATIONS: As specific brain structures are associated with stress, drug treatments targeting these areas (ie, gamma-aminobutyric acid-ergic drugs, serotonin and norepinephrine reuptake inhibitors) may help to modulate itch.	gamma-Aminobutyric Acid	114-137	itchy E3 ubiquitin protein ligase	Homo sapiens	222-226	
31940043-4	2020	Spinal inhibitory interneurons release GABA, glycine and dynorphin to modulate segmental itch transmission.	gamma-Aminobutyric Acid	39-43	itchy E3 ubiquitin protein ligase	Homo sapiens	89-93	
31000426-4	2019	Here, we report that GABAergic (GABA) and dopaminergic (DA) neurons in the ventral tegmental area (VTA) are activated with different temporal patterns during acute and chronic itch.	gamma-Aminobutyric Acid	21-25	itchy E3 ubiquitin protein ligase	Homo sapiens	176-180	
31000426-6	2019	Optogenetic manipulations of VTA GABA neurons rapidly modulated scratching behaviors through encoding itch-associated aversion.	gamma-Aminobutyric Acid	33-37	itchy E3 ubiquitin protein ligase	Homo sapiens	102-106