PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19799583-2	2010	Corticosterone is proposed to modulate cocaine intravenous self-administration (SA) and cocaine-induced locomotion through distinct receptors, the glucocorticoid receptor (GR) and the mineralocorticoid receptor (MR), respectively.	Cocaine	88-95	nuclear receptor subfamily 3, group C, member 2	Mus musculus	184-210	
19799583-2	2010	Corticosterone is proposed to modulate cocaine intravenous self-administration (SA) and cocaine-induced locomotion through distinct receptors, the glucocorticoid receptor (GR) and the mineralocorticoid receptor (MR), respectively.	Cocaine	88-95	nuclear receptor subfamily 3, group C, member 2	Mus musculus	212-214	
19799583-6	2010	Our goal here was to test the involvement of the GR and the MR in cocaine-induced locomotor and reinforcing effects in the same experimental conditions.	Cocaine	66-73	nuclear receptor subfamily 3, group C, member 2	Mus musculus	60-62	
19799583-12	2010	A blockade of the MR decreased both cocaine-induced reinforcing (but to a much lesser extent than the GR blockade) and locomotor effects.	Cocaine	36-43	nuclear receptor subfamily 3, group C, member 2	Mus musculus	18-20	
19799583-13	2010	Altogether, our results comforted the hypothesis that the GR modulates cocaine-related operant conditioning, while the MR would modulate cocaine-related unconditioned effects.	Cocaine	137-144	nuclear receptor subfamily 3, group C, member 2	Mus musculus	119-121