PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17488862-1	2007	BACKGROUND: Cocaine is a sympathomimetic agent that can cause coronary artery vasospasm leading to myocardial ischaemia, acute coronary syndrome and acute myocardial infarction (ACS/AMI).	Cocaine	12-19	1-aminocyclopropane-1-carboxylate synthase homolog (inactive)	Homo sapiens	178-181	
17488862-2	2007	The management of cocaine-induced ACS/AMI is different to classical atheromatous ACS/MI, because the mechanisms are different.	Cocaine	18-25	1-aminocyclopropane-1-carboxylate synthase homolog (inactive)	Homo sapiens	34-37	
17488862-6	2007	Retrospective study--Cocaine use or non-use was documented in 3.7% (4/109) and 4% (2/50) of clinical notes of patients with suspected and proven ACS, respectively.	Cocaine	21-28	1-aminocyclopropane-1-carboxylate synthase homolog (inactive)	Homo sapiens	145-148	
17488862-7	2007	DISCUSSION: Although junior medical staff are aware that cocaine is a risk factor for ACS/AMI, they are less likely to ask about it in routine clinical practice or record its use/non-use in clinical notes.	Cocaine	57-64	1-aminocyclopropane-1-carboxylate synthase homolog (inactive)	Homo sapiens	86-93	
17488862-8	2007	It is essential that patients presenting with suspected ACS are asked about cocaine use, since the management of these patients is different to those with ACS secondary to "classical" cardiovascular risk factors.	Cocaine	76-83	1-aminocyclopropane-1-carboxylate synthase homolog (inactive)	Homo sapiens	56-59