PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30711191-4	2019	We also assessed the moderating influences of participant age, sex, and IQ on our findings and the correlations of rCBF with N-acetylaspartate metabolite levels.	N-acetylaspartate	125-142	CCAAT/enhancer binding protein zeta	Rattus norvegicus	115-119	
30711191-8	2019	rCBF in the ASD group correlated inversely with N-acetylaspartate metabolite levels throughout the frontal white matter, with greater rCBF accompanying lower and increasingly abnormal N-acetylaspartate levels relative to those of typically developing control subjects.	N-acetylaspartate	48-65	CCAAT/enhancer binding protein zeta	Rattus norvegicus	0-4	
30711191-8	2019	rCBF in the ASD group correlated inversely with N-acetylaspartate metabolite levels throughout the frontal white matter, with greater rCBF accompanying lower and increasingly abnormal N-acetylaspartate levels relative to those of typically developing control subjects.	N-acetylaspartate	184-201	CCAAT/enhancer binding protein zeta	Rattus norvegicus	0-4	
11988598-8	2002	There was a tendency for larger rCBF defect size to be associated with greater increases in Cr/NAA values in the same diaschitic cerebral hemisphere, ipsilateral to the infarction.	N-acetylaspartate	95-98	CCAAT/enhancer binding protein zeta	Rattus norvegicus	32-36	
11988598-10	2002	The tendency for greater reductions in cortical rCBF values to be associated with increased Cr/NAA values in the same diaschitic cerebral hemisphere implies that a relationship exists between rCBF reductions in gray matter and abnormal changes in white matter subservient to it.	N-acetylaspartate	95-98	CCAAT/enhancer binding protein zeta	Rattus norvegicus	48-52	
11988598-10	2002	The tendency for greater reductions in cortical rCBF values to be associated with increased Cr/NAA values in the same diaschitic cerebral hemisphere implies that a relationship exists between rCBF reductions in gray matter and abnormal changes in white matter subservient to it.	N-acetylaspartate	95-98	CCAAT/enhancer binding protein zeta	Rattus norvegicus	192-196	
34762311-7	2021	Path analyses showed that reduced NAA levels accounted significantly for higher diffusivity and higher rCBF values in ASD compared with TD participants.	N-acetylaspartate	34-37	CCAAT/enhancer binding protein zeta	Rattus norvegicus	103-107