PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31376479-8	2019	Novel molecular analyses from stages before the onset of motor deficits revealed a strong selective effect of ATXN2 on Nat8l mRNA which encodes the enzyme responsible for NAA synthesis.	N-acetylaspartate	171-174	ataxin 2	Homo sapiens	110-115	
23094053-4	2012	V-NAA and patient age were used to calculate the predicted age at which a patient with SCA2 or SCA3 would reach an onset V-NAA value.	N-acetylaspartate	2-5	ataxin 2	Homo sapiens	87-91	
23094053-4	2012	V-NAA and patient age were used to calculate the predicted age at which a patient with SCA2 or SCA3 would reach an onset V-NAA value.	N-acetylaspartate	123-126	ataxin 2	Homo sapiens	87-91	
15240431-7	2004	Compared with control subjects, the SCA1 and SCA2 patients showed a decrease (P < 0.01) in the volume of the brainstem and cerebellum and in the concentration of NAA in the pons and cerebellar hemisphere, whereas D of the brainstem and cerebellum was increased.	N-acetylaspartate	165-168	ataxin 2	Homo sapiens	45-49	
15240431-2	2004	Magnetic resonance (MR) techniques enable in vivo measurement of the atrophy of the brainstem and cerebellum in spinocerebellar ataxia type 1 (SCA1) and 2 (SCA2) patients, which is accompanied by a decrease in the concentration of N-acetyl aspartate (NAA) or of the NAA/creatine ratio in the pons and cerebellum.	N-acetylaspartate	231-249	ataxin 2	Homo sapiens	156-160	
15240431-2	2004	Magnetic resonance (MR) techniques enable in vivo measurement of the atrophy of the brainstem and cerebellum in spinocerebellar ataxia type 1 (SCA1) and 2 (SCA2) patients, which is accompanied by a decrease in the concentration of N-acetyl aspartate (NAA) or of the NAA/creatine ratio in the pons and cerebellum.	N-acetylaspartate	251-254	ataxin 2	Homo sapiens	156-160	
15240431-2	2004	Magnetic resonance (MR) techniques enable in vivo measurement of the atrophy of the brainstem and cerebellum in spinocerebellar ataxia type 1 (SCA1) and 2 (SCA2) patients, which is accompanied by a decrease in the concentration of N-acetyl aspartate (NAA) or of the NAA/creatine ratio in the pons and cerebellum.	N-acetylaspartate	266-269	ataxin 2	Homo sapiens	156-160