PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18040781-2	2006	SIV infection and CD8 depletion resulted in a rapid decline in cerebral N-acetylaspartate (NAA) levels, a sensitive marker of neuronal health.	N-acetylaspartate	72-89	CD8a molecule	Homo sapiens	18-21	
18040781-2	2006	SIV infection and CD8 depletion resulted in a rapid decline in cerebral N-acetylaspartate (NAA) levels, a sensitive marker of neuronal health.	N-acetylaspartate	91-94	CD8a molecule	Homo sapiens	18-21	
18040781-8	2006	The rapid decline in NAA/Cr with SIV infection/CD8 depletion and its rapid recovery with HAART suggest that: (1) infected monocyte turnover in the CNS is rapid, occurring in days to weeks; (2) there are endogenous mechanisms that reverse neuronal injury; and (3) a threshold level of infected monocytes/macrophages in the CNS is required to overcome the neuronal recovery processes.	N-acetylaspartate	21-24	CD8a molecule	Homo sapiens	47-50	
26959509-7	2016	The CD4/CD8 ratio at baseline correlated with N-acetyl-aspartate (r = 0.56, P = 0.003) and choline (r = 0.36, P = 0.03) at 5 years, irrespective of treatment regimen and ART interruption.	N-acetylaspartate	46-64	CD8a molecule	Homo sapiens	8-11