PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15352223-6	2004	In concordance with the ex vivo data, the in vivo spectra of late-stage patients with CLN1 (n = 3) revealed a dramatic decrease of NAA and a proportional increase of myo-inositol and lipids compared with control subjects.	N-acetylaspartate	131-134	palmitoyl-protein thioesterase 1	Homo sapiens	86-90	
15352223-4	2004	Principal component analysis of ex vivo data showed that decreased levels of N-acetylaspartate (NAA), gamma-aminobutyric acid (GABA), glutamine, and glutamate as well as increased levels of inositols characterized the CLN1 spectra.	N-acetylaspartate	77-94	palmitoyl-protein thioesterase 1	Homo sapiens	218-222	
26339674-8	2015	RESULTS: In INCL patients, N-acetylaspartate (NAA) was abnormally low at all locations upon initial measurement, and further declined throughout the follow-up period.	N-acetylaspartate	27-44	palmitoyl-protein thioesterase 1	Homo sapiens	12-16	
26339674-8	2015	RESULTS: In INCL patients, N-acetylaspartate (NAA) was abnormally low at all locations upon initial measurement, and further declined throughout the follow-up period.	N-acetylaspartate	46-49	palmitoyl-protein thioesterase 1	Homo sapiens	12-16