PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32187825-3	2020	Using smooth muscle cells (VSMCs) from mouse mesenteric arteries, we found that both Cav 3.2 channel inhibition by Ni2+ (50 microM) and caveolae disruption by methyl-ss-cyclodextrin or genetic abolition of Eps15 homology domain-containing protein (EHD2) inhibited Ca2+ sparks in cells from young (4 months) but not old (12 months) mice.	Cyclodextrins	169-181	calcium channel, voltage-dependent, T type, alpha 1H subunit	Mus musculus	85-92