PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17584982-5	2007	We demonstrated that the LPS induced TNF-alpha response is in direct correlation with the biophase level of NE, as it is significantly higher when the release of NE of vesicular origin was completely inhibited in an animal model of depression (reserpine treatment) and it is significantly lower in the case of increasing biophase levels of NE by genetic (NET-KO) or chemical (desipramine) disruption of NE reuptake.	Desipramine	376-387	tumor necrosis factor	Mus musculus	37-46	
27571014-12	2016	The ischemia-induced increase in retinal TNF-alpha levels was suppressed by the administration of the ASMase inhibitor desipramine, or by reducing ASMase expression.	Desipramine	119-130	tumor necrosis factor	Mus musculus	41-50	
21867719-12	2012	and desipramine (16 mg/kg, i.p) prevented the depressant-like effect induced by TNF-alpha (0.1 fg/site, i.c.v.)	Desipramine	4-15	tumor necrosis factor	Mus musculus	80-89	
17584982-6	2007	IL-10 was changed inversely to TNF-alpha levels only in the desipramine-treated animals.	Desipramine	60-71	tumor necrosis factor	Mus musculus	31-40	
12782200-6	2003	Acute treatment of both wild-type and NET-KO mice with desipramine significantly decreased the TNF-alpha response and significantly increased interleukin-10 production, indicating the role of an intact noradrenaline transporter in anti-inflammatory responses.	Desipramine	55-66	tumor necrosis factor	Mus musculus	95-104