PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2615927-3	1989	The main findings were (1) sex differences in the growth hormone and cortisol response to desipramine and (2) a significant genetic component of the prolactin and cortisol response to desipramine as indicated by significantly (p less than 0.05) lower within-pair than between-pair variance in the sibling pairs but not random pairs of the experimental subjects.	Desipramine	184-195	prolactin	Homo sapiens	149-158	
2101962-0	1990	Influence of clenbuterol, a beta-adrenergic agonist, on desipramine induced growth hormone, prolactin and cortisol stimulation.	Desipramine	56-67	prolactin	Homo sapiens	92-101	
4001280-2	1985	In previous studies it was shown that the tricyclic antidepressant desimipramine (DMI) had different stimulatory effects on growth hormone (GH), prolactin (PRL), ACTH and cortisol secretion in healthy subjects, depending on the mode of administration.	Desipramine	67-80	prolactin	Homo sapiens	145-154	
3031717-2	1986	In this report the effects of various receptor blockers on desimipramine (DMI)-induced prolactin (PRL) secretion in healthy male subjects are presented.	Desipramine	59-72	prolactin	Homo sapiens	87-96	
3031717-2	1986	In this report the effects of various receptor blockers on desimipramine (DMI)-induced prolactin (PRL) secretion in healthy male subjects are presented.	Desipramine	74-77	prolactin	Homo sapiens	87-96	
4001280-2	1985	In previous studies it was shown that the tricyclic antidepressant desimipramine (DMI) had different stimulatory effects on growth hormone (GH), prolactin (PRL), ACTH and cortisol secretion in healthy subjects, depending on the mode of administration.	Desipramine	67-80	prolactin	Homo sapiens	156-159	
4001280-2	1985	In previous studies it was shown that the tricyclic antidepressant desimipramine (DMI) had different stimulatory effects on growth hormone (GH), prolactin (PRL), ACTH and cortisol secretion in healthy subjects, depending on the mode of administration.	Desipramine	82-85	prolactin	Homo sapiens	145-154	
4001280-2	1985	In previous studies it was shown that the tricyclic antidepressant desimipramine (DMI) had different stimulatory effects on growth hormone (GH), prolactin (PRL), ACTH and cortisol secretion in healthy subjects, depending on the mode of administration.	Desipramine	82-85	prolactin	Homo sapiens	156-159	
4001280-6	1985	Compared to placebo, significant increases occurred in GH (p less than 0.05) and in PRL (p less than 0.05) from a dose of DMI 25 mg on, and in cortisol (p less than 0.05) from 15 mg on.	Desipramine	122-125	prolactin	Homo sapiens	84-87	
6703855-4	1984	Both desipramine (N = 7) and amitriptyline (N = 6) significantly increased the PRL rise induced by tryptophan compared with a preceding placebo period.	Desipramine	5-16	prolactin	Homo sapiens	79-82	
6098913-5	1984	DMI acutely increased plasma PRL, whereas ZIM had an effect only after chronic pretreatment.	Desipramine	0-3	prolactin	Homo sapiens	29-32	
6098913-6	1984	Chronic DMI but not ZIM increased baseline PRL.	Desipramine	8-11	prolactin	Homo sapiens	43-46	
6703855-5	1984	In contrast, following long-term amitriptyline and desipramine treatment, the ability of tryptophan to increase PRL was enhanced two weeks following abrupt cessation of amitriptyline therapy (N = 5), but not after discontinuation of desipramine therapy.	Desipramine	51-62	prolactin	Homo sapiens	112-115	
6703855-5	1984	In contrast, following long-term amitriptyline and desipramine treatment, the ability of tryptophan to increase PRL was enhanced two weeks following abrupt cessation of amitriptyline therapy (N = 5), but not after discontinuation of desipramine therapy.	Desipramine	233-244	prolactin	Homo sapiens	112-115	
6420832-0	1984	Comparison of growth hormone and prolactin stimulation induced by chlorimipramine and desimipramine in man in connection with chlorimipramine metabolism.	Desipramine	86-99	prolactin	Homo sapiens	33-42	
582390-6	1978	The increase in PRL and TSH, which is induced by the thyrotrophin releasing hormone (TRH), is provable after acute as well as after chronic administration of DMI.	Desipramine	158-161	prolactin	Homo sapiens	16-19	
6124090-10	1982	It is likely that such studies will enrich our understanding of how these agents work, of the difference between agents which have been classed together on the basis of preclinical studies (e.g., DMI, which appears to increase PRL and GH, and NT which appears not to) and provided additional evidence to test current hypotheses about the biological basis of their antidepressant action.	Desipramine	196-199	prolactin	Homo sapiens	227-230