PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9314045-2	1997	We have investigated the effects of long-term treatment with desipramine on the phosphorylation state of microtubule-associated protein 2 (MAP2) and microtubule assembly in the rat cerebral cortex.	Desipramine	61-72	microtubule-associated protein 2	Rattus norvegicus	105-137	
9314045-2	1997	We have investigated the effects of long-term treatment with desipramine on the phosphorylation state of microtubule-associated protein 2 (MAP2) and microtubule assembly in the rat cerebral cortex.	Desipramine	61-72	microtubule-associated protein 2	Rattus norvegicus	139-143	
9314045-4	1997	The degree of phosphorylation of serine residues of MAP2 was significantly increased after chronic administration of desipramine without changes in the total concentration of MAP2.	Desipramine	117-128	microtubule-associated protein 2	Rattus norvegicus	52-56	
9314045-8	1997	These results raise the possibility that the changes in the degree of phosphorylation of MAP2 and microtubule assembly represent intracellular modifications involved in functional changes elicited by long-term treatment with desipramine.	Desipramine	225-236	microtubule-associated protein 2	Rattus norvegicus	89-93	
2550266-8	1989	Under our conditions the maximal effect occurred at 1 microM cAMP, revealing increased 32P incorporation in microtubule-associated protein 2 from a crude microtubule preparation obtained from the cerebral cortex of rats treated with desmethylimipramine.	Desipramine	233-252	microtubule-associated protein 2	Rattus norvegicus	108-140	
8564731-7	1995	The serine but not threonine or tyrosine phosphorylation of MAP2 was significantly increased after chronic treatment with DMI, MPR or CTR.	Desipramine	122-125	microtubule-associated protein 2	Rattus norvegicus	60-64