PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11093763-3 2000 N-(3-Trifluoromethylphenyl)piperazine, a 5-HT(1B) receptor agonist, potently inhibited 5-HT(1A) receptor-mediated slow inhibitory postsynaptic potentials (IPSPs) in the dorsal raphe of wild-type but not knockout mice. 1-(3-trifluoromethylphenyl)piperazine 0-37 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 41-58 9291149-0 1997 TFMPP, a 5HT1B receptor agonist, inhibits light-induced phase shifts of the circadian activity rhythm and c-Fos expression in the mouse suprachiasmatic nucleus. 1-(3-trifluoromethylphenyl)piperazine 0-5 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 9-23 10234032-5 1999 1-[3-(Trifluoromethyl)phenyl]-piperazine HCl (TFMPP), a 5-HT1B receptor agonist, reduced in a dose-related manner the amplitude of glutamatergic EPSCs evoked by stimulating selectively the optic nerve. 1-(3-trifluoromethylphenyl)piperazine 46-51 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 56-71 9291149-4 1997 We tested this hypothesis using the 5HT1B receptor agonist, 1-[3-(trifluoromethyl)phenyl]-piperazine (TFMPP). 1-(3-trifluoromethylphenyl)piperazine 60-100 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 36-50 9291149-4 1997 We tested this hypothesis using the 5HT1B receptor agonist, 1-[3-(trifluoromethyl)phenyl]-piperazine (TFMPP). 1-(3-trifluoromethylphenyl)piperazine 102-107 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 36-50 1628159-13 1992 In the social behaviour deficit test, anpirtoline and trifluoromethylphenyl-piperazine were effective in reversing the isolation-induced impairments in mice, an effect shown only by compounds with agonist properties at the 5-HT1B receptor. 1-(3-trifluoromethylphenyl)piperazine 54-86 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 223-238 9169822-4 1997 In addition, topical administration of the 5-HT1B agonist M-trifluoromethylphenylpiperazine (2 x 10(-6) to 2 x 10(-4) M) produced opposite responses, i.e., dose-dependent vasodilation in TIA and control arterioles, and dose-dependent constriction in TFA. 1-(3-trifluoromethylphenyl)piperazine 58-91 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 43-49 7972302-2 1994 The behavioral model used (increase in escape attempts of isolated mice) has been previously shown to be exclusively responsive to 5-HT1B agonists such as 1-3-(trifluoromethyl) phenylpiperazine (TFMPP). 1-(3-trifluoromethylphenyl)piperazine 155-193 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 131-137 7972302-2 1994 The behavioral model used (increase in escape attempts of isolated mice) has been previously shown to be exclusively responsive to 5-HT1B agonists such as 1-3-(trifluoromethyl) phenylpiperazine (TFMPP). 1-(3-trifluoromethylphenyl)piperazine 195-200 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 131-137 1971720-1 1990 Seven days of isolation induce in mice a social behavioral deficit (decrease in escape attempts) reversed by TFMPP acting through activation of 5-HT1B receptors. 1-(3-trifluoromethylphenyl)piperazine 109-114 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 144-150 1833660-1 1991 In mice, isolation-induced social behavioural deficits are attenuated by stimulants of 5-HT1B receptors, such as TFMPP or CGS 120 66B. 1-(3-trifluoromethylphenyl)piperazine 113-118 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 87-93 1975081-6 1990 The preferential 5-HT1B agents TFMPP and mCPP exhibited a profile similar to PTZ. 1-(3-trifluoromethylphenyl)piperazine 31-36 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 17-23 1975081-8 1990 The decreased entries and time spent on the open arm of the maze following TFMPP or mCPP administration was possibly mediated by an antagonistic action at 5-HT1B receptors, since this effect was reversed by the selective 5-HT1B agonist CGS 12066B. 1-(3-trifluoromethylphenyl)piperazine 75-80 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 155-161 1975081-8 1990 The decreased entries and time spent on the open arm of the maze following TFMPP or mCPP administration was possibly mediated by an antagonistic action at 5-HT1B receptors, since this effect was reversed by the selective 5-HT1B agonist CGS 12066B. 1-(3-trifluoromethylphenyl)piperazine 75-80 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 221-227 2969949-0 1988 Hypothermia induced by m-trifluoromethylphenylpiperazine or m-chlorophenylpiperazine: an effect mediated by 5-HT1B receptors? 1-(3-trifluoromethylphenyl)piperazine 23-56 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 108-114 3252258-1 1988 The effect of TFMPP, an agonist of the 5-HT1b receptors, was studied in mice on several psychopharmacological parameters. 1-(3-trifluoromethylphenyl)piperazine 14-19 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 39-45 3252258-6 1988 It is concluded that TFMPP seems to possess psychotropic activity resembling only in part that of imipramine-like drugs and that these actions may be mediated through 5-HT1b receptors. 1-(3-trifluoromethylphenyl)piperazine 21-26 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 167-173 2969949-6 1988 The obtained results indicate that the TFMPP- and m-CPP-induced hypothermias in mice are mediated by 5-HT1B. 1-(3-trifluoromethylphenyl)piperazine 39-44 5-hydroxytryptamine (serotonin) receptor 1B Mus musculus 101-107